Home Dynamics and prevention of gemini virus infection in red chili crops studied with generalized fractional operator: Analysis and modeling
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Dynamics and prevention of gemini virus infection in red chili crops studied with generalized fractional operator: Analysis and modeling

  • Kottakkaran Sooppy Nisar EMAIL logo , Muhammad Farman and Fahad Sameer Alshammari
Published/Copyright: September 24, 2025
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Abstract

The gemini virus, a major obstacle to red chili production, is exacerbated by yellow virus propagation. This study explores the potential of an epidemic model using generalized fractal fractional operators to observe dynamics and develop prevention strategies to control infections. The fractional-ordering system is analyzed quantitatively and qualitatively, including positiveness, boundedness, unique solution, and reproductive analysis under equilibrium points to ensure bounded and positive solutions. The proposed model’s uniqueness is demonstrated through global effects analysis using Lipschitz and linear growth techniques, and local and global stability was assessed using the Lyapunov function and the first derivative test. The study utilizes a two-level Lagrange polynomial, specifically the Mittag–Leffler kernel, to explore the impact of fractional operators on plant diseases. The fractional-order model’s behavior is verified through numerical simulations at disease-free and equilibrium points, and results are compared to demonstrate its efficacy and memory effect. The study visually illustrates the impact of various proposed operators on the proposed red chilli model, providing numerical data for each operator with varying fractional parameters. By comparing non-integer orders to integer orders, we obtain a more comparable result to support its stance. The study found that the fractal fractional operator is more effective than the usual integer order for disease eradication because it efficiently reduces gemini virus infection rates by lowering the fractional-order parameter ϑ . This study will allow us to develop mitigating techniques for afflicted plants and gain a better understanding of the virus’s behavior.

1 Introduction

An important agricultural crop in Indonesia is red chili (Capsicum annuum). Being one of the most expensive agricultural crops, C. annuum can improve farmers’ income in addition to being used as a vegetable or cooking spice [14]. The call for C. annuum is rising year after year after 12 months in Indonesia, along with the population increase and the increase of industries that use uncooked materials from C. annuum. This is a result of pest troubles and plant diseases that reduce productivity [57]. The gemini virus, a yellow virus that could damage C. annuum and is spread through insect vectors, is one of the problems in the manufacturing of this plant. Human can contract the disease by sucking on an infected C. annuum, and after it has propagated throughout his body. Stunted, yellow, and curved (cupping) leaves are only a few of the symptoms of this viral disease [2,3, 8,9]. Insect pathogenic fungus (Verticillium lecanii) can be used to solve this issue. For suppressing Bemisia tabaci, the entomopathogen V. lecanii is a useful fungus. V. lecanii has recently gained widespread use as a pesticide substitute [1012]. V. lecanii is applied to C. annuum by misting suspensions on the underside of the leaves [13]. However, if V. lecanii is used excessively, the growth of the chilli plant may be inhibited. It takes further information to conduct a more detailed examination of the effectiveness of using V. lecanii. A mathematical model is employed by one of them [14].

Numerous scientists have created mathematical models for plant diseases, some of which take into account the relationship between the vector and the host in disease transmission [1517]. Shi and colleagues [18] examined the mathematical prey–predator model in connection with the host–vector theory. Seema Khekare and Sujatha Janardhan [19] examined a host–vector epidemic model with non-linear and bilinear events. Anggriani et al. [20] developed a mathematical model of plant disease by taking both preventative and remedial measures into account, examined the use of insecticides to control Tungro disease vectors [21], and examined the application of fungicides for both prevention and treatment [22]. Wang et al. [23] have examined how the age of infection and worldwide prevalence rates affect models of vector-borne disease dynamics. Researchers [24,25] reported on how to best manage fungicide effects. The possibility of microbe-based biological control as environmentally benign substitutes for chemical pesticides in chili growing is assessed in the study by Sam On et al. [26]. Chemical pesticides are extremely dangerous for both the environment and human health. The research highlights the significance of sustainable disease control strategies, host–pathogen interaction, and integrated disease management approaches [27]. Recent research has shown RNA structures that impact virus replication and need to migrate from cell to cell to enter nearby cells [28]. Previous studies [29,30] list some recent developments and potential paths forward.

Studying mathematical models with fractional derivatives rather than regular derivatives produces more meaningful findings and facilitates comprehension of challenging issues. In the literature, a wide range of fractional-order derivatives have been introduced and applied. Numerous real-world applications [3136] of fractional calculus exist. Convolutions are used to define fractional derivatives; as a specific instance, they contain ordinary derivatives. Due to its many attributes and realistic solutions to problems in engineering and physics, fractional calculus is attracting the interest of researchers everywhere on the globe. The simplest model with a fractional-order device lets in for the observation of hereditary characteristics, memory, and crossover conduct [37,38]. There is a new approach to the fractal fractional derivative in fractional calculus, and it was recently developed by Atangana [39]. The core ideas in this area are well suited to dealing with complex problems in numerous contexts. This new approach to calculating fractal fractions outperforms the standard approach by a significant margin [4043]. The significant benefit of this operator is that it facilitates the development of models that more adequately account for the occurrence of memory effects in complex systems. Furthermore, there are other practical concerns where understanding the information capacity of a system is essential. Researchers suggested a few developments in fractal-fractional differential equations, making use of novel programs and diverse kernels [4446].

This study uses a fractal-fractional mathematical model to investigate the transmission of yellow viruses in red chilli plants, focusing on the dynamics of yellow virus transmission in C. annuum, a plant disease that has gained significant attention in recent years. The rest of this article is organized as follows: The main important factors of red chili plant viruses with vector transmission disorders are covered in Section 2, along with the fractional-order red chili plant virus version. We demonstrate the bondedness and positiveness effects. We discuss the implications of global derivatives. Results are developed for strong points and lifestyles. Reproduction range, equilibrium factors, and local and global stability analyses with Lyapunov characteristic are all evaluated in Section 3; a numerical scheme using the Newton polynomial is given in Section 4; we discuss the results of the simulation in Section 5 and the findings in Section 6.

2 Fractional-order model of the spread of the yellow virus in red chili plants

Recent research indicates that fractional calculus and fractional-order model methodologies have greatly enhanced real-world processes in science and engineering. This has resulted in the use of classical identification techniques to these fields. The flexibility of fractional differential equations, which can work in any order, benefits first- and lower-order models alike. These transfer function models are critical for recognizing real-time behavioral characteristics.

Definition 2.1

[48] The Mittag–Leffler function is defined as follows:

(1) E ϑ = n = 0 t n Γ ( ϑ n + 1 ) .

Definition 2.2

[49] For a function g H 1 ( 0 , C ) and 0 < ϑ 1 , the Atangana–Baleanu–Caputo (ABC) derivative can be defined as

(2) D t ϑ 0 A B C [ g ( t ) ] = A B ( ϑ ) 1 ϑ 0 t g ( υ ) E ϑ ϑ ( t υ ) ϑ 1 ϑ d υ ,

where the space of square-integrable functions, H 1 ( 0 , C ) , with C > 0 , is defined as follows:

(3) H 1 ( 0 , C ) = { g ( t ) L 2 ( 0 , C ) f ( t ) L 2 ( 0 , C ) } ,

and A B ( ϑ ) = 1 ϑ + ϑ Γ ( ϑ ) with A B ( 0 ) = A B ( 1 ) = 1 .

The corresponding ABC integral can be defined as

(4) I t ϑ 0 A B C [ g ( t ) ] = 1 ϑ A B ( ϑ ) g ( t ) + ϑ A B ( ϑ ) Γ ( ϑ ) 0 t g ( υ ) ( t υ ) ϑ 1 d υ .

Atangana [39] developed a new class of fractional concepts for rationalizing systems by integrating fractal derivatives with power-law, exponential law, and modified Mittag–Leffler equation. This research looks into a unique fractal-like starting value problem employing a fractal-fractional operator with a Mittag–Leffler kernel.

Definition 2.3

[39,43,50] Consider g : ( a , b ) [ 0 , ) as a continuous, fractal differentiable map with dimension ζ . The fractal-fractional derivative of g with a generalized Mittag–Leffler kernel of order ϑ is as follows:

(5) D t ϑ , ζ 0 F F M [ g ( t ) ] = A B ( ϑ ) 1 ϑ d d t ζ 0 t g ( υ ) E ϑ ϑ ( t υ ) ϑ 1 ϑ d υ ,

where E ϑ is the Mittag–Leffler kernel and the fractal derivative is given by

(6) d g ( υ ) d υ ζ = lim t υ g ( t ) g ( υ ) t ζ υ ζ .

The corresponding integral is as follows:

(7) I t ϑ , ζ 0 F F M [ g ( t ) ] = ( 1 ϑ ) ζ t ζ 1 A B ( ϑ ) g ( t ) + ϑ ζ A B ( ϑ ) Γ ( ϑ ) 0 t υ ζ 1 ( t υ ) ϑ 1 g ( υ ) d υ .

We present deterministic compartmental model [47] of yellow virus in red chili plants as a deeper study of the data is required to determine the spread of the yellow virus in the red chili plant, and one method is using mathematical modeling. The population of red chilli plants is divided into four classes:

  • Red chilli plants that are susceptible in the vegetative stage ( S v ( t ) ) ;

  • Red chilli plants with infection in the vegetative stage ( I v ( t ) ) ;

  • Red chilli plants that are susceptible in the generative stage ( S g ( t ) ) ;

  • Red chilli plants with infection in the generative stage ( I g ( t ) ) ;

Also, there are two classes of insect vectors:
  • Susceptible insects ( S B T ( t ) ) ;

  • Infected insects ( I B T ( t ) ) .

The total population of red chili plants and insects vectors are denoted by

N α = S v + I v + S g + I g , N β = S B T + I B T .

Here, we present a fractional-order model for the spread of the yellow virus in red chili plants in the modified form:

(8) D t ϑ , ζ 0 F F M S v ( t ) = C λ S v μ 1 ( 1 σ p ) S v I B T ψ p S v , D t ϑ , ζ 0 F F M I v ( t ) = μ 1 ( 1 σ p ) S v I B T ψ p I v , D t ϑ , ζ 0 F F M S g ( t ) = λ S v μ 2 ( 1 σ p ) S g I B T ψ p S g , D t ϑ , ζ 0 F F M I g ( t ) = μ 2 ( 1 σ p ) S g I B T ψ p I g , D t ϑ , ζ 0 F F M S B T ( t ) = E N β ν 1 ( 1 σ p ) I v S B T ν 2 ( 1 σ p ) I g S B T φ I σ p S B T N α ψ I S B T , D t ϑ , ζ 0 F F M I B T ( t ) = ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T φ I σ p I B T N α ψ I I B T ,

with initial conditions (Table 1)

(9) S v ( 0 ) 0 , I v ( 0 ) 0 , S p ( 0 ) 0 , I p ( 0 ) 0 , S B T ( 0 ) 0 , I B T ( 0 ) 0 .

Table 1

Parameters of the recommended model

N α The total population of red chili plants
N β The total population of insect vectors (B. tabaci)
C Population recruitment of red chili plants
E N β Population recruitment of B. tabaci
λ The rate of plant growth when it moves from the vegetative phase to the generative phase
μ 1 ( 1 σ p ) The efficiency of applying V. lecanii affects how frequently plants become infected during the vegetative period
μ 2 ( 1 σ p ) The efficiency of applying V. lecanii affects how frequently plants become infected during the generative period
ν 1 ( 1 σ p ) The efficiency of using V. lecanii influences the rate of B. tabaci infection when infected red chilli plants are interacted with during the vegetative stage
ν 2 ( 1 σ p ) The efficiency of using V. lecanii influences the rate of B. tabaci infection when infected red chilli plants are interacted with during the generative stage
σ p Use of V. lecanii effectiveness
ψ p Red chilli plant mortality rates
ψ I The proportion of B. tabaci natural deaths
φ I The success of V. lecanii when applied to red chilli plants
σ p The mortality rate of B. tabaci

3 Proposed model analysis

3.1 Positive bounded solutions

For the model’s population description to be valid, the state parameters must be non-negative for any t > 0 . The solution should be limited and positive for all time values greater than or equal to zero if the original data are positive. To prevent negative values in a population system, it is necessary to demonstrate the positivity of solutions and maintain the positive beginning value in feasible region. Positivity and boundedness are critical features for population systems, with boundedness preferred due to resource constraints.

3.1.1 Positiveness

Theorem 3.1

[50] Suppose that t 0 . For model (8), if the initial conditions fulfill

S v ( 0 ) , I v ( 0 ) , S g ( 0 ) , I g ( 0 ) , S B T ( 0 ) , I B T ( 0 ) > 0 ,

then S v ( t ) , I v ( t ) , S g ( t ) , I g ( t ) , S B T ( t ) , and I B T ( t ) will remain positive in R + 6 .

Proof

The basic analysis discusses conditions that guarantee the positivity of the proposed model’s solutions and seeks to ascertain the preferability of solutions based on their actual difficulties with positive values. First, we start from I v ( t ) class

(10) D t ϑ , ζ 0 F F M I v ( t ) ψ p I v t 0 I v ( t ) I V ( 0 ) E ϑ c 1 ζ ϑ ( ψ p ) t ϑ A B ( ϑ ) ( 1 ϑ ) ( ψ p ) , t 0 ,

where c represents a time component. We shall define the norm

(11) θ = sup t D θ θ ( t ) ,

where D θ is the domain of θ . We have

(12) D t ϑ , ζ 0 F F M S g ( t ) ( μ 2 ( 1 σ p ) sup t D I B T I B T ψ p ) S g ( t ) , t 0 ( μ 2 ( 1 σ p ) I B T ψ p ) S g ( t ) , t 0 S g ( t ) S g ( 0 ) E ϑ c 1 ζ ϑ ( μ 2 ( 1 σ p ) I B T + ψ p ) t ϑ A B ( ϑ ) ( 1 ϑ ) ( μ 2 ( 1 σ p ) I B T + ψ p ) , t 0 ,

(13) D t ϑ , ζ 0 F F M I g ( t ) ψ p I g , t 0 I g ( t ) I g ( 0 ) E ϑ c 1 ζ ϑ ( ψ p ) t ϑ A B ( ϑ ) ( 1 ϑ ) ( ψ p ) , t 0 .

D t ϑ , ζ 0 F F M S B T ( t ) ( E ν 1 ( 1 σ p ) sup t D I v I v ν 2 ( 1 σ p ) sup t D I g I g φ I σ p sup t D N α N α ψ I ) S B T , t 0 ( E + ν 1 ( 1 σ p ) I v + ν 2 ( 1 σ p ) I g + φ I σ p N α + ψ I ) S B T , t 0 .

(14) S B T ( t ) S B T ( 0 ) E ϑ c 1 ζ ϑ ( E + ν 1 ( 1 σ p ) I v + ν 2 ( 1 σ p ) I g + φ I σ p N α + ψ I ) t ϑ A B ( ϑ ) ( 1 ϑ ) ( ν 1 ( 1 σ p ) I v E + ν 2 ( 1 σ p ) I g + φ I σ p N α + ψ I ) , t 0 .

(15) D t ϑ , ζ 0 F F M I B T ( t ) ( φ I σ p sup t D N α N α ψ I ) I B T , t 0 ( φ I σ p N α + ψ I ) I B T , t 0 I B T ( t ) I B T ( 0 ) E ϑ c 1 ζ ϑ ( φ I σ p N α + ψ I ) t ϑ A B ( ϑ ) ( 1 ϑ ) ( φ I σ p N α + ψ I ) , t 0 .

(16) D t ϑ , ζ 0 F F M S v ( t ) ( λ μ 1 ( 1 σ p ) sup t D I B T I B T ψ p ) S v , t 0 ( λ + μ 1 ( 1 σ p ) I B T + ψ p ) S v t 0 S v ( t ) S v ( 0 ) E ϑ c 1 ζ ϑ ( λ + μ 1 ( 1 σ p ) I B T + ψ p ) t ϑ A B ( ϑ ) ( 1 ϑ ) ( λ + μ 1 ( 1 σ p ) I B T + ψ p ) , t 0 .

This implies that I v ( t ) , S g ( t ) , I g ( t ) , S B T ( t ) , I B T ( t ) , and S v ( t ) are all positive for all t 0 .□

Theorem 3.2

[43] Along with initial conditions, the proposed plant virus model’s solution is unique and limited in R + 6 .

Proof

We obtain

(17) D t ϑ , ζ 0 F F M ( S v ( t ) ) S v = 0 = C 0 , D t ϑ , ζ 0 F F M ( I v ( t ) ) I v = 0 = μ 1 ( 1 σ p ) S v I B T 0 , D t ϑ , ζ 0 F F M ( S g ( t ) ) S g = 0 = λ S v 0 , D t ϑ , ζ 0 F F M ( I g ( t ) ) I g = 0 = μ 2 ( 1 σ p ) S g I B T 0 , D t ϑ , ζ 0 F F M ( S B T ( t ) ) S B T = 0 = E I B T 0 , D t ϑ , ζ 0 F F M ( I B T ( t ) ) I B T = 0 = ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T 0 .

Since ( S v ( 0 ) , I v ( 0 ) , S g ( 0 ) , I g ( 0 ) , S B T ( 0 ) , I B T ( 0 ) ) R + 6 , the solution cannot escape from the hyperplanes of S v ( t ) = 0 , I v ( t ) = 0 , S g ( t ) = 0 , I g ( t ) = 0 , S B T ( t ) = 0 , and I B T ( t ) = 0 , and the vector field on each hyperplane is either tangent to that hyperplane or points toward the interior of domain R + 6 ; i.e., since the solution will stay in the domain R + 6 , this domain is a positive invariant set.□

3.1.2 Boundedness

Theorem 3.3

With positive initial conditions, every solution of model (8)is bounded [50].

Proof

Theorems 3.1 and 3.2 ensure the positivity of solutions; hence,

D t ϑ , ζ 0 F F M N α = C ψ p N α N α = C ψ p + N α ( 0 ) C ψ p E ϑ c 1 ζ ϑ ( ψ p ) t ϑ A B ( ϑ ) ( 1 ϑ ) ( ψ p ) M 1 ,

where M 1 = max { N α ( 0 ) , C ψ p } and

D t ϑ , ζ 0 F F M N β = ( E φ I σ p N α ψ I ) N β N β = N β ( 0 ) E ϑ c 1 ζ ϑ ( E φ I σ p N α ψ I ) t ϑ A B ( ϑ ) ( 1 ϑ ) ( E φ I σ p N α ψ I ) N β ( 0 ) .

The solutions are bounded to the region Ξ , where

Ξ = { S v ( t ) , I v ( t ) , S g ( t ) , I g ( t ) , S B T ( t ) , I B T ( t ) R + 6 N α M 1 = max N α ( 0 ) , C ψ p , N β N β ( 0 ) , t 0 .

It demonstrates that for any t 0 , all solutions remain positive invariant under the specified beginning conditions in region Ξ .□

3.2 Well-posedness and biological feasibility

Here, we determine the range and area in which the model’s solution makes perfect sense. All solutions are positive, as demonstrated by Theorems 3.1 and 3.2, which also provide suggested parameters for all t 0 . Theorem 3.3 further demonstrates the boundedness of the suggested model:

(18) Ξ = { ( S v ( t ) , I v ( t ) , S g ( t ) , I g ( t ) , S B T ( t ) , I B T ( t ) ) R + 6 : 0 N α M 1 = max N α ( 0 ) , C ψ p : 0 N β N β ( 0 ) } ,

where R + 6 represents the positive cone of R 6 in biological terms that also include its lower-dimensional faces.

3.3 Global derivative’s effect

With an emphasis on proportionate fractional definitions and compartments, this section presents a compartmental model using fractional calculus. To account for the uncertainty of disease transmission, the model is expanded to a stochastic extension and decimated to integer order. It can create standards to identify unique and constructive global solutions. Although research into qualitative fractional calculus theories is restricted, the existence of solutions to many models has pushed researchers to accelerate their efforts. They investigated epidemic models and proposed the fractional model qualitative theory, which will be discussed in this work. It has long been known in the literature that the Riemann–Stieltjes integral, of which the classical integral is a special example, is the most often occurring integral. When

F ( x ) = f ( x ) d x ,

the Riemann–Stieltjes integral is given by

F g ( x ) = f ( x ) d g ( x ) .

f ( x ) ’s global derivative in terms of g ( x ) is given by

D g f ( x ) = lim h 0 f ( x + h ) f ( x ) g ( x + h ) g ( x ) .

If classical differentiation is possible for both functions, then

D g f ( x ) = f ( x ) g ( x ) , g ( x ) 0 , x D g .

We will replace the global derivative with the classical derivative to see if it affects the plant virus model:

(19) D g S v ( t ) = C λ S v μ 1 ( 1 σ p ) S v I B T ψ p S v , D g I v ( t ) = μ 1 ( 1 σ p ) S v I B T ψ p I v , D g S g ( t ) = λ S v μ 2 ( 1 σ p ) S g I B T ψ p S g , D g I g ( t ) = μ 2 ( 1 σ p ) S g I B T ψ p I g , D g S B T ( t ) = E N β ν 1 ( 1 σ p ) I v S B T ν 2 ( 1 σ p ) I g S B T φ I σ p S B T N α ψ I S B T , D g I B T ( t ) = ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T φ I σ p I B T N α ψ I I B T .

We shall assume that g is differentiable for simplicity’s sake. Consequently,

(20) S v ( t ) = g [ C λ S v μ 1 ( 1 σ p ) S v I B T ψ p S v ] , I v ( t ) = g [ μ 1 ( 1 σ p ) S v I B T ψ p I v ] , S g ( t ) = g [ λ S v μ 2 ( 1 σ p ) S g I B T ψ p S g ] , I g ( t ) = g [ μ 2 ( 1 σ p ) S g I B T ψ p I g ] , S B T ( t ) = g [ E N β ν 1 ( 1 σ p ) I v S B T ν 2 ( 1 σ p ) I g S B T φ I σ p S B T N α ψ I S B T ] , I B T ( t ) = g [ ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T φ I σ p I B T N α ψ I I B T ] .

Let

(21) g = sup t D g g ( t ) < N .

This example will demonstrate that there is a single solution that can be accepted by the system of equations:

(22) S v ( t ) = g [ C λ S v μ 1 ( 1 σ p ) S v I B T ψ p S v ] = Z 1 Θ , I v ( t ) = g [ μ 1 ( 1 σ p ) S v I B T ψ p I v ] = Z 2 Θ , S g ( t ) = g [ λ S v μ 2 ( 1 σ p ) S g I B T ψ p S g ] = Z 3 Θ , I g ( t ) = g [ μ 2 ( 1 σ p ) S g I B T ψ p I g ] = Z 4 Θ , S B T ( t ) = g [ E N β ν 1 ( 1 σ p ) I v S B T ν 2 ( 1 σ p ) I g S B T φ I σ p S B T N α ψ I S B T ] = Z 5 Θ , I B T ( t ) = g [ ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T φ I σ p I B T N α ψ I I B T ] = Z 6 Θ .

Here, we look at the system’s equations that support the existence and uniqueness of fractional calculus, which requires the proof of the following:

  1. Z Θ 2 < κ ( 1 + S v 2 ) ,

  2. S v 1 , S v 2 , we have,

    Z ( t , S v 1 , I v , S g , I g , S B T , I B T ) Z ( t , S v 2 , I v , S g , I g , S B T , I B T ) 2 < κ ¯ S v 1 S v 2 2 ,

where Θ = ( t , S v , I v , S g , I g , S B T , I B T ) . Consider Θ 1 = ( t , S v 1 , I v , S g , I g , S B T , I B T ) and Θ 2 = ( t , S v 2 , I v , S g , I g , S B T , I B T ) and if

Z 1 ( Θ 1 ) Z 1 ( Θ 2 ) 2 = g ( λ μ 1 ( 1 σ p ) I B T ψ p ) ( S v 1 S v 2 ) 2 Z 1 ( Θ 1 ) Z 1 ( Θ 2 ) 2 g 2 ( 3 λ 2 + 3 ( μ 1 ( 1 σ p ) ) 2 I B T 2 + 3 ( ψ p ) 2 ) S v 1 S v 2 2 sup t D S v Z 1 ( Θ 1 ) Z 1 ( Θ 2 ) 2 sup t D g g 2 ( 3 λ 2 + 3 ( μ 1 ( 1 σ p ) ) 2 sup t D I B T I B T 2 + 3 ( ψ p ) 2 ) sup t D S v S v 1 S v 2 2 Z 1 ( Θ 1 ) Z 1 ( Θ 2 ) 2 g 2 ( 3 λ 2 + 3 ( μ 1 ( 1 σ p ) ) 2 I B T 2 + 3 ( ψ p ) 2 ) S v 1 S v 2 2 Z 1 ( Θ 1 ) Z 1 ( Θ 2 ) 2 κ ¯ 1 S v 1 S v 2 2 ,

where

κ ¯ 1 = g 2 ( 3 λ 2 + 3 ( μ 1 ( 1 σ p ) ) 2 I B T 2 + 3 ( ψ p ) 2 ) .

Now consider Θ 3 = ( t , S v , I v 1 , S g , I g , S B T , I B T ) and Θ 4 = ( t , S v , I v 2 , S g , I g , S B T , I B T ) and if

Z 2 ( Θ 3 ) Z 2 ( Θ 4 ) 2 = g ( ψ p ) ( I v 1 I v 2 ) 2 , Z 2 ( Θ 3 ) Z 2 ( Θ 4 ) 2 g 2 ( ψ p ) 2 ( I v 1 I v 2 ) 2 , sup t D I v Z 2 ( Θ 3 ) Z 2 ( Θ 4 ) 2 sup t D g g 2 ( ψ p ) 2 sup t D I v ( I v 1 I v 2 ) 2 , Z 2 ( Θ 3 ) Z 2 ( Θ 4 ) 2 g 2 ( ψ p ) 2 ( I v 1 I v 2 ) 2 , Z 2 ( Θ 3 ) Z 2 ( Θ 4 ) 2 κ ¯ 2 ( I v 1 I v 2 ) 2 ,

where

κ ¯ 2 = g 2 ( ψ p ) 2 .

Now, consider Θ 5 = ( t , S v , I v , S g 1 , I g , S B T , I B T ) and Θ 6 = ( t , S v , I v , S g 1 , I g , S B T , I B T ) and if

Z 3 ( Θ 5 ) Z 3 ( Θ 6 ) 2 = g ( μ 2 ( 1 p ) I B T ψ p ) ( S g 1 S g 2 ) 2 , Z 3 ( Θ 5 ) Z 3 ( Θ 6 ) 2 g 2 ( 2 ( μ 2 ( 1 p ) ) 2 I B T 2 + 2 ( ψ p ) 2 ) ( S g 1 S g 2 ) 2 , sup t D S g Z 3 ( Θ 5 ) Z 3 ( Θ 6 ) 2 sup t D g g 2 ( 2 ( μ 2 ( 1 p ) ) 2 sup t D I B T I B T 2 + 2 ( ψ p ) 2 ) sup t D S g ( S g 1 S g 2 ) 2 , Z 3 ( Θ 5 ) Z 3 ( Θ 6 ) 2 g 2 ( 2 ( μ 2 ( 1 p ) ) 2 I B T 2 + 2 ( ψ p ) 2 ) ( S g 1 S g 2 ) 2 , Z 3 ( Θ 5 ) Z 3 ( Θ 6 ) 2 κ ¯ 3 ( S g 1 S g 2 ) 2 ,

where

κ ¯ 3 = g 2 ( 2 ( μ 2 ( 1 p ) ) 2 I B T 2 + 2 ( ψ p ) 2 ) .

Now, consider Θ 7 = ( t , S v , I v , S g , I g 1 , S B T , I B T ) and Θ 8 = ( t , S v , I v , S g , I g 2 , S B T , I B T ) and if

Z 4 ( Θ 7 ) Z 2 ( Θ 8 ) 2 = g ( ψ p ) ( I g 1 I g 2 ) 2 , Z 4 ( Θ 7 ) Z 2 ( Θ 8 ) 2 g 2 ( ψ p ) 2 ( I g 1 I g 2 ) 2 , sup t D I v Z 4 ( Θ 7 ) Z 2 ( Θ 8 ) 2 sup t D g g 2 ( ψ p ) 2 sup t D I v ( I g 1 I g 2 ) 2 , Z 4 ( Θ 7 ) Z 2 ( Θ 8 ) 2 g 2 ( ψ p ) 2 ( I g 1 I g 2 ) 2 , Z 4 ( Θ 7 ) Z 2 ( Θ 8 ) 2 κ ¯ 4 ( I g 1 I g 2 ) 2 ,

where

κ ¯ 4 = g 2 ( ψ p ) 2 .

Now, consider Θ 9 = ( t , S v , I v , S g , I g , S B T 1 , I B T ) and Θ 10 = ( t , S v , I v , S g , I g , S B T 2 , I B T ) and if

Z 5 ( Θ 9 ) Z 5 ( Θ 10 ) 2 = g ( ν 1 ( 1 σ p ) I v ν 2 ( 1 σ p ) I g φ I σ p N α ψ I ) ( S g 1 S g 2 ) 2 Z 5 ( Θ 9 ) Z 5 ( Θ 10 ) 2 4 g 2 ( ( 1 σ p ) 2 ( ν 1 2 I v 2 + ν 2 2 I g 2 ) + ( φ I σ p ) 2 N α 2 + ( ψ I ) 2 ) ( S B T 1 S B T 2 ) 2 sup t D S B T Z 5 ( Θ 9 ) Z 5 ( Θ 10 ) 2 4 sup t D g g 2 ( ( 1 σ p ) 2 ( ν 1 2 sup t D I v I v 2 + ν 2 2 ) sup t D I g I g 2 + ( φ I σ p ) 2 sup t D N α N α 2 + ( ψ I ) 2 ) sup t D S B T ( S B T 1 S B T 2 ) 2 Z 5 ( Θ 9 ) Z 5 ( Θ 10 ) 2 4 g 2 ( ( 1 σ p ) 2 ( ν 1 2 I v 2 + ν 2 2 I g 2 ) + ( φ I σ p ) 2 N α 2 + ψ I 2 ) ( S B T 1 S B T 2 ) 2 κ ¯ 5 ( S B T 1 S B T 2 ) 2 ,

where

κ ¯ 5 = g 2 ( 4 ( ν 1 ( 1 σ p ) ) 2 I v 2 + 4 ( ν 2 ( 1 σ p ) ) 2 I g 2 + 4 ( φ I σ p ) 2 N α 2 + 4 ( ψ I ) 2 ) .

Now, consider Θ 11 = ( t , S v , I v , S g , I g , S B T , I B T 1 ) and Θ 12 = ( t , S v , I v , S g , I g , S B T , I B T 2 ) and if

Z 6 ( Θ 11 ) Z 6 ( Θ 12 ) 2 = g ( φ I σ p N α ψ I ) ( I B T 1 I B T 2 ) 2 Z 6 ( Θ 11 ) Z 6 ( Θ 12 ) 2 2 g 2 ( ( φ I σ p ) 2 N α 2 + ψ I 2 ) ( I B T 1 I B T 2 ) 2 sup t D I B T Z 6 ( Θ 11 ) Z 6 ( Θ 12 ) 2 2 sup t D g g 2 ( ( φ I σ p ) 2 sup t D N α N α 2 + ψ I 2 ) sup t D I B T ( I B T 1 I B T 2 ) 2 Z 6 ( Θ 11 ) Z 6 ( Θ 12 ) 2 2 g 2 ( ( φ I σ p ) 2 N α 2 + ψ I 2 ) ( I B T 1 I B T 2 ) 2 Z 6 ( Θ 11 ) Z 6 ( Θ 12 ) 2 κ ¯ 6 ( I B T 1 I B T 2 ) 2 ,

where

κ ¯ 6 = g 2 ( 2 ( φ I σ p ) 2 N α 2 + 2 ( ψ I ) 2 ) .

This demonstrates that function satisfies the first requirement. We then confirm that the second criterion is met:

Z 1 ( Θ ) 2 = g [ C λ S v μ 1 ( 1 σ p ) S v I B T ψ p S v ] 2 2 g 2 C 2 + 2 g 2 λ S v μ 1 ( 1 σ p ) S v I B T ψ p S v 2 2 g 2 C 2 + 6 g 2 λ 2 S v 2 + 6 g 2 ( μ 1 ( 1 σ p ) ) 2 S v 2 I B T 2 + 6 g 2 ψ p 2 S v 2 2 g 2 C 2 1 + ( 6 λ 2 + 6 ( μ 1 ( 1 σ p ) ) 2 I B T 2 + 6 ψ p 2 ) S v 2 2 C 2 κ 1 ( 1 + S v 2 ) ,

under the condition

( 6 λ 2 + 6 ( μ 1 ( 1 σ p ) ) 2 I B T 2 + 6 ψ p 2 ) 2 C 2 < 1 ,

where

κ 1 = 2 g 2 C 2 .

Z 2 ( Θ ) 2 = g [ μ 1 ( 1 σ p ) S v I B T ψ p I v ] 2 2 g 2 μ 1 ( 1 σ p ) S v I B T 2 + 2 g 2 ψ p I v 2 2 g 2 μ 1 2 ( 1 σ p ) 2 S v 2 I B T 2 + 2 g 2 ψ p 2 I v 2 2 g 2 μ 1 2 ( 1 σ p ) 2 S v 2 I B T 2 1 + 2 ψ p 2 I v 2 2 μ 1 2 ( 1 σ p ) 2 S v 2 I B T 2 κ 2 ( 1 + I v 2 ) ,

under the condition

2 ψ p 2 2 μ 1 2 ( 1 σ p ) 2 S v 2 I B T 2 < 1 ,

where

κ 2 = 2 g 2 μ 1 2 ( 1 σ p ) 2 S v 2 I B T 2 .

Z 3 ( Θ ) 2 = g [ λ S v μ 2 ( 1 σ p ) S g I B T ψ p S g ] 2 2 g 2 λ S v 2 + 2 g 2 μ 2 ( 1 σ p ) S g I B T ψ p S g 2 2 g 2 λ 2 S v 2 + 4 g 2 μ 2 2 ( 1 σ p ) 2 S g 2 I B T 2 + 4 g 2 ψ p 2 S g 2 2 g 2 λ 2 S v 2 1 + 4 ( μ 2 2 ( 1 σ p ) 2 I B T 2 + 4 ψ p 2 ) S g 2 2 S v 2 κ 3 ( 1 + S g 2 ) ,

under the condition

4 ( μ 2 2 ( 1 σ p ) 2 I B T 2 + 4 ψ p 2 ) 2 S v 2 < 1 ,

where

κ 3 = 2 g 2 S v 2 .

Z 4 ( Θ ) 2 = g [ μ 2 ( 1 σ p ) S g I B T ψ p I g ] 2 2 g 2 μ 2 ( 1 σ p ) S g I B T 2 + 2 g 2 ψ p I g 2 2 g 2 μ 2 2 ( 1 σ p ) 2 S g 2 I B T 2 + 2 g 2 ψ p 2 I g 2 2 g 2 μ 2 2 ( 1 σ p ) 2 S g 2 I B T 2 ( 1 + f r a c 2 ψ p 2 I g 2 2 μ 2 2 ( 1 σ p ) 2 S g 2 I B T 2 ) κ 4 ( 1 + I g 2 ) ,

under the condition

2 ψ p 2 2 μ 2 2 ( 1 σ p ) 2 S g 2 I B T 2 < 1 ,

where

κ 4 = 2 g 2 μ 2 2 ( 1 σ p ) 2 S g 2 I B T 2 .

Z 5 ( Θ ) 2 = g [ E N β ν 1 ( 1 σ p ) I v S B T ν 2 ( 1 σ p ) I g S B T φ I σ p S B T N α ψ I S B T ] 2 2 g 2 [ E N β 2 + ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T + φ I σ p S B T N α + ψ I S B T 2 ] 2 g 2 E 2 N β 2 + 8 g 2 [ ( ν 1 ( 1 σ p ) ) 2 I v 2 + ( ν 2 ( 1 σ p ) ) 2 I g 2 + ( φ I σ p ) 2 N α 2 + ( ψ I ) 2 ] S B T 2 2 g 2 E 2 N β 2 × 1 + 8 [ ( ν 1 ( 1 σ p ) ) 2 I v 2 + ( ν 2 ( 1 σ p ) ) 2 I g 2 + ( φ I σ p ) 2 N α 2 + ( ψ I ) 2 ] S B T 2 2 E 2 N β 2 κ 5 ( 1 + S B T 2 ) ,

under the condition

8 [ ( ν 1 ( 1 σ p ) ) 2 I v 2 + ( ν 2 ( 1 σ p ) ) 2 I g 2 + ( φ I σ p ) 2 N α 2 + ( ψ I ) 2 ] 2 E 2 N β 2 < 1 ,

where

κ 5 = 2 g 2 E 2 N β 2 .

Z 6 ( Θ ) 2 = g [ ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T φ I σ p I B T N α ψ I I B T ] 2 2 g 2 ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T 2 + 2 g 2 φ I σ p I B T N α ψ I I B T 2 4 g 2 ( ν 1 2 ( 1 σ p ) 2 I v 2 S B T 2 + ν 2 2 ( 1 σ p ) 2 I g 2 S B T 2 + φ I 2 σ p 2 I B T 2 N α 2 + ψ I 2 I B T 2 ) 4 g 2 ( ( 1 σ p ) 2 ( ν 1 2 I v 2 S B T 2 + 4 ν 2 2 I g 2 S B T 2 ) ) 1 + 4 φ I 2 σ p 2 I B T 2 N α 2 + 4 ψ I 2 I B T 2 4 ( 1 σ p ) 2 ( ν 1 2 I v 2 S B T 2 + ν 2 2 I g 2 S B T 2 ) κ 6 ( 1 + I B T 2 ) ,

under the condition

4 φ I 2 σ p 2 I B T 2 N α 2 + 4 ψ I 2 4 ν 1 2 ( 1 σ p ) 2 I v 2 S B T 2 + 4 ν 2 2 ( 1 σ p ) 2 I g 2 S B T 2 < 1 ,

where

κ 6 = 4 g 2 ν 1 2 ( 1 σ p ) 2 I v 2 S B T 2 + 4 ν 2 2 ( 1 σ p ) 2 I g 2 S B T 2 .

As a result, our system has a solution that is unique given the following circumstances:

(23) Max 2 ψ p 2 2 μ 1 2 ( 1 σ p ) 2 S v 2 I B T 2 , 2 ψ p 2 2 μ 1 2 ( 1 σ p ) 2 S v 2 I B T 2 , 4 ( μ 2 2 ( 1 σ p ) 2 I B T 2 + 4 ψ p 2 ) 2 S v 2 , 2 ψ p 2 2 μ 2 2 ( 1 σ p ) 2 S g 2 I B T 2 , 8 [ ( ν 1 ( 1 σ p ) ) 2 I v 2 + ( ν 2 ( 1 σ p ) ) 2 I g 2 + ( φ I σ p ) 2 N α 2 + ( ψ I ) 2 ] 2 E 2 N β 2 , 4 φ I 2 σ p 2 I B T 2 N α 2 + 4 ψ I 2 4 ν 1 2 ( 1 σ p ) 2 I v 2 S B T 2 + 4 ν 2 2 ( 1 σ p ) 2 I g 2 S B T 2 . < 1

3.4 Equilibrium points

In order to determine equilibrium points, we must set the system’s equations (8) to zero. The disease-free equilibrium point is obtained from model [8] as follows:

(24) E 0 ( S v 1 , I v 1 , S g 1 , I g 1 , S B T 1 , I B T 1 ) = C ψ p + λ , 0 , C λ ψ p + λ , 0 , E N β φ I σ p N α + ψ I , 0 .

The endemic equilibrium is given by E 1 ( S v * , I v * , S g * , I g * , S B T * , I B T * ) , where

(25) S v * = C ( ψ p + λ ) + μ 1 ( 1 σ p ) I B T * , I v * = μ 1 ( 1 σ p ) S v * I B T * ψ p , S g * = λ S v * μ 2 ( 1 σ p ) I B T * + ψ p , I g * = μ 2 ( 1 σ p ) S g * I B T * ψ p , S B T * = E N β ν 1 ( 1 σ p ) I v * + ν 2 ( 1 σ p ) I g * + φ I σ p N α + ψ I ,

and I B T * , unique positive solution of the following equation is given as

(26) h ( I B T * ) = a ( I B T * ) 2 + b I B T * + c ,

where

(27) a = μ 1 μ 2 ( 1 σ p ) 2 ( φ I σ p N α + ψ I ) ( C ν 1 ( 1 σ p ) + ( φ I σ p N α + ψ I ) ψ p ) > 0 , b = ( 1 σ p ) { [ ( φ I σ p N α + ψ I ) 2 ψ p ( ψ p ( μ 1 + μ 2 ) + λ μ 2 ) ] + C ( 1 σ p ) [ ( ψ p μ 1 ν 1 + ψ p μ 2 ν 2 ) ( φ I σ p N α + ψ I ) + E N β μ 1 μ 2 ν 1 ( 1 σ p ) ] } > 0 , c = ( 1 R 0 2 ) < 0 .

3.5 Reproduction number

To continue with our prior analysis, here we shall derive the reproductive number. Reproduction number plays an important role in the field of epidemiological modeling, as it helps to understand the stability conditions. The vectors F and V stand in for the genesis of new illnesses and the spread of already-existing infections, respectively. Let the system be

(28) D t ϑ , ζ 0 F F M I v ( t ) = μ 1 ( 1 σ p ) S v I B T ψ p I v , D t ϑ , ζ 0 F F M I g ( t ) = μ 2 ( 1 σ p ) S g I B T ψ p I g , D t ϑ , ζ 0 F F M I B T ( t ) = ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T φ I σ p I B T N α ψ I I B T .

Then,

F = μ 1 ( 1 σ p ) S v I B T μ 2 ( 1 σ p ) S g I B T ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T , V = ψ p I v ψ p I g φ I σ p I B T N p + ψ I I B T .

The Jacobian matrices of F and V are found at E 0 . We then have

F = 0 0 C μ 1 ( 1 σ p ) ψ p + λ 0 0 C λ μ 2 ( 1 σ p ) ψ p + λ E N β ν 1 ( 1 σ p ) φ I σ p N α + ψ I E N β ν 2 ( 1 σ p ) φ I σ p N α + ψ I 0 and V = ψ p 0 0 0 ψ p 0 0 0 C φ I σ p + ψ I ψ p ψ p

F V 1 = 0 0 C ψ p μ 1 ( 1 σ p ) ( ψ p + λ ) ( φ I σ p C + ψ I ψ p ) 0 0 C λ μ 2 ( 1 σ p ) ( ψ p + λ ) ( φ I σ p C + ψ I ψ p ) E N β ν 1 ( 1 σ p ) ψ p ( φ I σ p N α + ψ I ) E N β ν 2 ( 1 σ p ) ψ p ( φ I σ p N α + ψ I ) 0

Thus, det ( F V 1 ξ I ) = ξ ξ 2 C E λ N β μ 2 ν 2 ( 1 σ p ) 2 ψ p ( ψ p + λ ) ( φ I σ p N α + ψ I ) ( φ I σ p C + ψ I ψ p ) + ξ C E N β μ 1 ν 1 ( 1 σ p ) 2 ( ψ p + λ ) ( φ I σ p N α + ψ I ) ( φ I σ p C + ψ I ψ p ) , where ξ 1 = 0 and ξ 2,3 = ± ( 1 σ p ) C E N β ( ψ p μ 1 ν 1 + λ μ 2 ν 2 ) ψ p ( φ I σ p N α + ψ I ) ( φ I σ p C + ψ I ψ p )

As the reproduction number is dominant eigenvalue of matrix F V 1 , so

(29) R 0 = R ( E 0 ) = ± ( 1 σ p ) C E N β ( ψ p μ 1 ν 1 + λ μ 2 ν 2 ) ψ p ( φ I σ p N α + ψ I ) ( φ I σ p C + ψ I ψ p ) .

The R 0 is the threshold frequency of infectious cases due to an infected specie, with the passage of time, in a red chilli plant population that is only susceptible. The population may get infected from virus when R 0 is greater than 1, but not when R 0 is less than 1.

3.6 Local stability analyses of equilibrium points

Here, we present theorems with proofs that describe local stability of equilibria.

Theorem 3.4

The disease-free equilibrium point of the proposed model is unstable if R 0 > 1 and locally asymptotically stable if R 0 < 1 [47].

Proof

System (8)’s Jacobian matrix at the disease-free equilibrium point is

J 0 = ψ p 0 C μ 1 ( σ p 1 ) ψ p + λ 0 0 0 0 ψ p C λ μ 2 ( σ p 1 ) ψ p ( ψ p + λ ) 0 0 0 E N β ν 1 ( σ p 1 ) ψ I + N α σ p φ I E N β ν 2 ( σ p 1 ) ψ I + N α σ p φ I C σ p φ I ψ p ψ I 0 0 0 0 C μ 1 ( σ p 1 ) ψ p + λ ( ψ p + λ ) 0 0 0 0 C λ μ 1 ( σ p 1 ) ψ p ( ψ p + λ ) λ ψ p 0 E N β ( ν 1 ( 1 σ p ) + σ p φ I ) ψ I + N α σ p φ I E N β ( ν 1 ( 1 σ p ) + σ p φ I ) ψ I + N α σ p φ I E ( E N β σ p φ I ) ψ I + N α σ p φ I ( E N β σ p φ I ) ψ I + N α σ p φ I E C σ p φ I ψ p ψ I

The disease free equilibrium is locally asymptotically stable if all eigenvalues ξ i , i = 1 , 2 , 3 , 4 of matrix J 0 satisfy condition

(30) arg ( eig ( J 0 ) ) = arg ( ξ i ) > υ Π 2 , i = 1 , 2 , 3 , 4 .

The characteristic equation is given as

( ( ψ p + ξ ) 2 ( ξ + λ + ψ p ) ( ξ + C σ p φ I + ( 1 ψ I E ) ψ p ) ( a ξ 2 + b ξ + c ) ) ψ p 2 ( ψ p + λ ) ( ψ I + σ p φ I N α ) = 0 ,

where

a = ψ p ( ψ p + λ ) ( σ p φ I N α + ψ I ) , b = ( ψ p + λ ) ( σ p φ I N α + ψ I ) ( C φ I σ p + ψ p ( ψ p + ψ I ) ) , c = ψ p ( ψ p + λ ) ( σ p φ I N α + ψ I ) ( C φ I σ p + ψ p ψ I ) C E N β ( ψ p μ 1 ν 1 + λ μ 2 ν 2 ) ( 1 σ p ) 2 ,

eigenvalues can be found out by solving the characteristic equation. Eigenvalues are given as

ξ 1,2 = ψ p , ξ 3 = ψ p λ , ξ 4 = ( C σ p φ I + ( ψ I E + 1 ) ψ p ) , ξ 5,6 = b ± b 2 4 a c 2 a .

From the aforementioned eigenvalues, it is clear that ξ 1,2,3 are negative. The system is stable if the values ξ 4,5,6 are also negative. To see this, consider the characteristic equation ( C σ p φ I + ( ψ I E + 1 ) ψ p ) ( a ξ 2 + b ξ + c ) = 0 , the system will be stable if the coefficients a, b, c and ( C σ p φ I + ( ψ I E + 1 ) ψ p ) are positive. So

a = ψ p ( ψ p + λ ) ( σ p φ I N α + ψ I ) > 0 , b = ( ψ p + λ ) ( σ p φ I N α + ψ I ) ( C φ I σ p + ψ p ( ψ p + ψ I ) ) > 0 , c = ψ p ( ψ p + λ ) ( σ p φ I N α + ψ I ) ( C φ I σ p + ψ p ψ I ) C E N β ( ψ p μ 1 ν 1 + λ μ 2 ν 2 ) ( 1 σ p ) 2 = ( 1 R 0 ) 2 > 0 .

From the coefficients for the characteristic equation ( a ξ 2 + b ξ + c ) = 0 , it can be seen that ξ 4,5,6 will be negative if E < C σ p φ I ψ p + ( ψ I + 1 ) and R 0 < 1 . This completes the proof.□

3.7 Global stability analysis

We now prove a few results related to global stability. One common Lyapunov candidate function for proving integer system stability is the quadratic function. Nevertheless, applying such functions is not simple in the fractional case. We put up the following lemma to prove the global stability of the fractional system (8).

Lemma 3.1

Let Z R + be a continuous function in which for each t t 0 ;

(31) D t ϑ , ζ 0 F F M Z Z Z ln Z Z 1 Z Z D t ϑ , ζ 0 F F M Z ( t ) , Z R + , ϑ , ζ ( 0 , 1 ) .

We define all independent variables for the Lyapunov function. For { S v 1 , I v 1 , S g 1 , I g 1 , S B T 1 , I B T 1 } , L < 0 is the endemic equilibrium.

Theorem 3.1

The endemic equilibrium points of a fractional-order red chilli plant’s system 8 are globally asymptotically stable if the reproductive number is greater than 1.

Proof

In this case, the Lyapunov function is defined as follows:

L ( S v * , I v * , S g * , I g * , S B T * , I B T * ) = l 1 S v S v * S v * log S v S v * + l 2 I v I v * I v * log I v I v * + l 3 S g S g * S g * log S g S g * + l 4 I g I g * I g * log I g I g * + l 5 S B T S B T * S B T * log S B T S B T * + l 6 I B T I B T * I B T * log I B T I B T * ,

where l 1 , l 2 , l 3 , l 4 , l 5 , and l 6 are the positive constants. By applying equation (32) to system (8), we obtain

(32) D t ϑ , ζ 0 F F M L S v S v * S v D t ϑ , ζ 0 F F M S v + I v I v * I v D t ϑ , ζ 0 F F M I v + S g S g * S g S g ˙ + I g I g * I g D t ϑ , ζ 0 F F M I g + S B T S B T * S B T D t ϑ , ζ 0 F F M S B T + I B T I B T * I B T D t ϑ , ζ 0 F F M I B T .

Writing their derivative values as

D t ϑ , ζ 0 F F M L S v S v * S v ( C λ S v μ 1 ( 1 σ p ) S v I B T ψ p S v ) + I v I v * I v ( μ 1 ( 1 σ p ) S v I B T ψ p I v ) + S g S g * S g ( λ S v μ 2 ( 1 σ p ) S g I B T ψ p S g ) + I g I g * I g ( μ 2 ( 1 σ p ) S g I B T ψ p I g ) + S B T S B T * S B T ( E N β ν 1 ( 1 σ p ) I v S B T ν 2 ( 1 σ p ) I g S B T φ I σ p S B T N α ψ I S B T ) + I B T I B T * I B T ( ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T φ I σ p I B T N α ψ I I B T ) .

Let S v = S v S v * , I v = I v I v * , S g = S g S g * , I g = I g I g * , S B T = S B T S B T * , I B T = I B T I B T * , then

D t ϑ , ζ 0 F F M L S v S v * S v ( C λ ( S v S v * ) μ 1 ( 1 σ p ) ( S v S v * ) ( I B T I B T * ) ψ p ( S v S v * ) ) + I v I v * I v ( μ 1 ( 1 σ p ) ( S v S v * ) ( I B T I B T * ) ψ p ( I v I v * ) ) + S g S g * S g ( λ ( S v S v * ) μ 2 ( 1 σ p ) ( S g S g * ) ( I B T I B T * ) ψ p ( I g I g * ) ) + I g I g * I g ( μ 2 ( 1 σ p ) ( S g S g * ) ( I B T I B T * ) ψ p ( S g S g * ) ) + S B T S B T * S B T ( E N β ν 1 ( 1 σ p ) ( I v I v * ) ( S B T S B T * ) ν 2 ( 1 σ p ) ( I g I g * ) ( S B T S B T * ) φ I σ p ( S B T S B T * ) N α ψ I ( S B T S B T * ) ) + I B T I B T * I B T ( ν 1 ( 1 σ p ) ( I v I v * ) ( S B T S B T * ) + ν 2 ( 1 σ p ) ( I g I g * ) ( S B T S B T * ) φ I σ p ( I B T I B T * ) N α ψ I ( I B T I B T * ) ) .

Using l 1 = l 2 = l 3 = l 4 = l 5 = l 6 = 1 and simplifying, we can write

(33) D t ϑ , ζ 0 F F M L Σ Ω ,

where

(34) Σ = C + μ 1 ( I B T * + σ p I B T ) ( S v S v * ) 2 S v + μ 1 ( S v I B T + S v * I B T * ) + μ 1 σ p ( S v I B T * + S v * I B T ) + μ 1 ( S v I B T * + S v * I B T ) I v * I v + μ 1 σ p ( S v I B T + S v * I B T * ) I v * I v + λ S v + λ S v * S g * S g + μ 2 ( I B T * + σ p I B T ) ( S g S g * ) 2 S g + ψ p I g * + ψ p I g S g * S g + μ 2 ( S g I B T + S g * I B T * ) + μ 2 σ p ( S g I B T * + S g * I B T ) + μ 2 ( S g I B T * + S g * I B T ) I g * I g + μ 2 σ p ( S g I B T + S g * I B T * ) I g * I g + ψ p S g * + ψ p S g I g * I g + E N β + ν 1 ( I v * σ p I v ) ( S B T S B T * ) 2 S B T + ν 2 ( I g * σ p I g ) ( S B T S B T * ) 2 S B T + ν 1 ( I v S B T + I v * S B T * ) + ν 1 ( I v S B T * + I v * S B T ) I B T * I B T + ν 2 ( I g S B T + I g * S B T * ) + ν 2 ( I g S B T * + I g * S B T ) I B T * I B T ,

and

(35) Ω = C S v * S v + λ ( S v S v * ) 2 S v + μ 1 ( I B T + σ p I B T * ) ( S v S v * ) 2 S v + ψ p ( S v S v * ) 2 S v + μ 1 ( S v I B T * + S v * I B T ) + μ 1 σ p ( S v I B T * + S v * I B T * ) + μ 1 ( S v I B T + S v * I B T * ) I v * I v + μ 1 σ p ( S v I B T * + S v * I B T ) I v * I v + ψ p ( I v I v * ) 2 I v + λ S v * + λ S v S g * S g + μ 2 ( I B T + σ p I B T * ) ( S g S g * ) 2 S g + ψ p I g + ψ p I g * S g * S g + μ 1 ( I B T + σ p I B T * ) ( S v S v * ) 2 S v + ψ p ( S v S v * ) 2 S v + μ 2 ( S g I B T * + S g * I B T ) + μ 2 σ p ( S g I B T * + S g * I B T * ) + μ 2 ( S g I B T + S g * I B T * ) I g * I g + μ 2 σ p ( S g I B T * + S g * I B T ) I g * I g + ψ p S g + ψ p S g * I g * I g + E N β S B T * S B T + ν 1 ( I v + σ p I v * ) S B T S B T * S B T + ν 2 ( I g + σ p I g * ) ( S B T S B T * ) 2 S B T + φ I σ p N α ( S B T S B T * ) 2 S B T + ψ I ( S B T S B T * ) 2 S B T + ν 1 ( I v S B T * + I v * S B T ) + ν 1 ( I v S B T + I v * S B T ) I B T * I B T + ν 1 ( I g S B T * + I g * S B T ) + ν 1 ( I g S B T + I g * S B T ) I B T * I B T + φ I σ p N α ( I B T I B T * ) 2 I B T + ψ I ( I B T I B T * ) 2 I B T .

We observe that

if Σ < Ω D t ϑ , ζ 0 F F M L < 0 .

However, if S v = S v * , I v = I v * , S g = S g * , I g = I g * , S B T = S B T * , I B T = I B T * then

(36) Σ Ω = 0 D t ϑ , ζ 0 F F M L = 0 .

As a result, we conclude that the endemic equilibrium points ( E * ) are globally asymptotically stable in feasible domain R + 6 if Σ < Ω .□

4 Numerical scheme

This section uses the generalized version of the fractal-fractional derivative in the Mittag–Leffler function to give a numerical algorithm for simulations and findings of the proposed system. Utilizing a Newton polynomial interpolation scheme [50], this research gives a numerical solution for interprets that shift from stretched exponential to power law without a steady-state and self-similarities. We can write model (8) as follows:

(37) D 0 ϑ , ζ 0 F F M S v ( t ) = S v 1 ( t , ϖ ) , D 0 ϑ , ζ 0 F F M I v ( t ) = I v 1 ( t , ϖ ) , D 0 ϑ , ζ 0 F F M S g ( t ) = S g 1 ( t , ϖ ) , D 0 ϑ , ζ 0 F F M I g ( t ) = I g 1 ( t , ϖ ) , D 0 ϑ , ζ 0 F F M S B T ( t ) = S B T 1 ( t , ϖ ) , D 0 ϑ , ζ 0 F F M I B T ( t ) = I B T 1 ( t , ϖ ) ,

where ϖ = S v , I v , S g , I g , S B T , I B T , and

(38) S v 1 ( t , ϖ ) = C λ S v μ 1 ( 1 σ p ) S v I B T ψ p S v , I v 1 ( t , ϖ ) = μ 1 ( 1 σ p ) S v I B T ψ p I v , S g 1 ( t , ϖ ) = λ S v μ 2 ( 1 σ p ) S g I B T ψ p S g , I g 1 ( t , ϖ ) = μ 2 ( 1 σ p ) S g I B T ψ p I g , S B T 1 ( t , ϖ ) = E N β ν 1 ( 1 σ p ) I v S B T ν 2 ( 1 σ p ) I g S B T φ I σ p S B T N α ψ I S B T , I B T 1 ( t , ϖ ) = ν 1 ( 1 σ p ) I v S B T + ν 2 ( 1 σ p ) I g S B T φ I σ p I B T N α ψ I I B T .

Applying integral (7), we obtain

(39) S v ( t δ + 1 ) = S v 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ S v 1 ( t δ , S v ( t δ ) , I v ( t δ ) , S g ( t δ ) , I g ( t δ ) , S B T ( t δ ) , I B T ( t δ ) ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ t o t o + 1 S v 1 ( τ , S v , I v , S g , I g , S B T , I B T ) τ 1 ζ ( t δ + 1 τ ) ϑ 1 d τ ,

(40) I v ( t δ + 1 ) = I v 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ I v 1 ( t δ , S v ( t δ ) , I v ( t δ ) , S g ( t δ ) , I g ( t δ ) , S B T ( t δ ) , I B T ( t δ ) ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ t o t o + 1 I v 1 ( τ , S v , I v , S g , I g , S B T , I B T ) τ 1 ζ ( t δ + 1 τ ) ϑ 1 d τ ,

(41) S g ( t δ + 1 ) = S g 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ S g 1 ( t δ , S v ( t δ ) , I v ( t δ ) , S g ( t δ ) , I g ( t δ ) , S B T ( t δ ) , I B T ( t δ ) ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ t o t o + 1 S g 1 ( τ , S v , I v , S g , I g , S B T , I B T ) τ 1 ζ ( t δ + 1 τ ) ϑ 1 d τ ,

(42) I g ( t δ + 1 ) = I g 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ I g 1 ( t δ , S v ( t δ ) , I v ( t δ ) , S g ( t δ ) , I g ( t δ ) , S B T ( t δ ) , I B T ( t δ ) ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ t o t o + 1 I g 1 ( τ , S v , I v , S g , I g , S B T , I B T ) τ 1 ζ ( t δ + 1 τ ) ϑ 1 d τ ,

(43) S B T ( t δ + 1 ) = S B T 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ S B T 1 ( t δ , S v ( t δ ) , I v ( t δ ) , S g ( t δ ) , I g ( t δ ) , S B T ( t δ ) , I B T ( t δ ) ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ t o t o + 1 S B T 1 ( τ , S v , I v , S g , I g , S B T , I B T ) τ 1 ζ ( t δ + 1 τ ) ϑ 1 d τ ,

(44) I B T ( t δ + 1 ) = I B T 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ I B T 1 ( t δ , S v ( t δ ) , I v ( t δ ) , S g ( t δ ) , I g ( t δ ) , S B T ( t δ ) , I B T ( t δ ) ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ t o t o + 1 I B T 1 ( τ , S v , I v , S g , I g , S B T , I B T ) τ 1 ζ ( t δ + 1 τ ) ϑ 1 d τ .

Substituting the Newton polynomial into the aforementioned equations, we have

(45) S v ( t δ + 1 ) = S v 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ S v 1 ( Ξ ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ S v 1 ( Ξ 3 ) t o 2 1 ζ t o t o + 1 ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 Δ t × [ t o 1 1 ζ S v 1 ( Ξ 2 ) t o 2 1 ζ S v 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ S v 1 ( Ξ 1 ) 2 t o 1 1 ζ S v 1 ( Ξ 2 ) + t o 2 1 η S v 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( τ t o 1 ) ( t δ + 1 τ ) ϑ 1 d τ ,

(46) I v ( t δ + 1 ) = I v 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ I v 1 ( Ξ ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ I v 1 ( Ξ 3 ) t o 2 1 ζ t o t o + 1 ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 Δ t [ t o 1 1 ζ I v 1 ( Ξ 2 ) t o 2 1 ζ I v 1 ( Ξ 3 ) ] × t o t o + 1 ( τ t o 2 ) ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ I v 1 ( Ξ 1 ) 2 t o 1 1 ζ I v 1 ( Ξ 2 ) + t o 2 1 η I v 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( τ t o 1 ) ( t δ + 1 τ ) ϑ 1 d τ ,

(47) S g ( t δ + 1 ) = S g 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ S g 1 ( Ξ ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ S g 1 ( Ξ 3 ) t o 2 1 ζ t o t o + 1 ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 Δ t × [ t o 1 1 ζ S g 1 ( Ξ 2 ) t o 2 1 ζ S g 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ S g 1 ( Ξ 1 ) 2 t o 1 1 ζ S g 1 ( Ξ 2 ) + t o 2 1 η S g 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( τ t o 1 ) ( t δ + 1 τ ) ϑ 1 d τ ,

(48) I g ( t δ + 1 ) = I g 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ I g 1 ( Ξ ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ I g 1 ( Ξ 3 ) t o 2 1 ζ t o t o + 1 ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 Δ t × [ t o 1 1 ζ I g 1 ( Ξ 2 ) t o 2 1 ζ I g 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ I g 1 ( Ξ 1 ) 2 t o 1 1 ζ I g 1 ( Ξ 2 ) + t o 2 1 η I g 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( τ t o 1 ) ( t δ + 1 τ ) ϑ 1 d τ ,

(49) S B T ( t δ + 1 ) = S B T 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ S B T 1 ( Ξ ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ S B T 1 ( Ξ 3 ) t o 2 1 ζ t o t o + 1 ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 Δ t [ t o 1 1 ζ S B T 1 ( Ξ 2 ) t o 2 1 ζ S B T 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ S B T 1 ( Ξ 1 ) 2 t o 1 1 ζ S B T 1 ( Ξ 2 ) + t o 2 1 η S B T 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( τ t o 1 ) ( t δ + 1 τ ) ϑ 1 d τ ,

(50) I B T ( t δ + 1 ) = I B T 0 + 1 ϑ A B ( ϑ ) t δ 1 ζ I B T 1 ( Ξ ) + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ I B T 1 ( Ξ 3 ) t o 2 1 ζ t o t o + 1 ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 Δ t [ t o 1 1 ζ I B T 1 ( Ξ 2 ) t o 2 1 ζ I B T 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( t δ + 1 τ ) ϑ 1 d τ + ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ I B T 1 ( Ξ 1 ) 2 t o 1 1 ζ I B T 1 ( Ξ 2 ) + t o 2 1 η I B T 1 ( Ξ 3 ) ] t o t o + 1 ( τ t o 2 ) ( τ t o 1 ) ( t δ + 1 τ ) ϑ 1 d τ .

After some calculations, we obtain finally (Figure 1)

(51) S v ( t δ + 1 ) = 1 ϑ A B ( ϑ ) t δ 1 ζ S v 1 ( Ξ ) + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 1 ) o = 2 δ S v 1 ( Ξ 3 ) t o 2 1 ζ × ϒ + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 2 ) o = 2 δ 1 Δ t × [ t o 1 1 ζ S v 1 ( Ξ 2 ) t o 2 1 ζ S v 1 ( Ξ 3 ) ] × Φ + ϑ ( Δ t ) ϑ 2 A B ( ϑ ) Γ ( ϑ + 3 ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ S v 1 ( Ξ 1 ) 2 t o 1 1 ζ S v 1 ( Ξ 2 ) + t o 2 1 η S v 1 ( Ξ 3 ) ] × Ψ ,

(52) I v ( t δ + 1 ) = 1 ϑ A B ( ϑ ) t δ 1 ζ I v 1 ( Ξ ) + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 1 ) o = 2 δ I v 1 ( Ξ 3 ) t o 2 1 ζ × ϒ + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 2 ) o = 2 δ 1 Δ t × [ t o 1 1 ζ I v 1 ( Ξ 2 ) t o 2 1 ζ I v 1 ( Ξ 3 ) ] × Φ + ϑ ( Δ t ) ϑ 2 A B ( ϑ ) Γ ( ϑ + 3 ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ I v 1 ( Ξ 1 ) 2 t o 1 1 ζ I v 1 ( Ξ 2 ) + t o 2 1 η I v 1 ( Ξ 3 ) ] × Ψ ,

(53) S g ( t δ + 1 ) = 1 ϑ A B ( ϑ ) t δ 1 ζ S g 1 ( Ξ ) + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 1 ) o = 2 δ S g 1 ( Ξ 3 ) t o 2 1 ζ × ϒ + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 2 ) o = 2 δ 1 Δ t × [ t o 1 1 ζ S g 1 ( Ξ 2 ) t o 2 1 ζ S g 1 ( Ξ 3 ) ] × Φ + ϑ ( Δ t ) ϑ 2 A B ( ϑ ) Γ ( ϑ + 3 ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ S g 1 ( Ξ 1 ) 2 t o 1 1 ζ S g 1 ( Ξ 2 ) + t o 2 1 η S g 1 ( Ξ 3 ) ] × Ψ ,

(54) I g ( t δ + 1 ) = 1 ϑ A B ( ϑ ) t δ 1 ζ I g 1 ( Ξ ) + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 1 ) o = 2 δ I g 1 ( Ξ 3 ) t o 2 1 ζ × ϒ + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 2 ) o = 2 δ 1 Δ t × [ t o 1 1 ζ I g 1 ( Ξ 2 ) t o 2 1 ζ I g 1 ( Ξ 3 ) ] × Φ + ϑ ( Δ t ) ϑ 2 A B ( ϑ ) Γ ( ϑ + 3 ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ I g 1 ( Ξ 1 ) 2 t o 1 1 ζ I g 1 ( Ξ 2 ) + t o 2 1 η I g 1 ( Ξ 3 ) ] × Ψ ,

(55) S B T ( t δ + 1 ) = 1 ϑ A B ( ϑ ) t δ 1 ζ S B T 1 ( Ξ ) + ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ S B T 1 ( Ξ 3 ) t o 2 1 ζ ϒ + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 2 ) o = 2 δ 1 Δ t [ t o 1 1 ζ S B T 1 ( Ξ 2 ) t o 2 1 ζ S B T 1 ( Ξ 3 ) ] Φ + ϑ ( Δ t ) ϑ 2 A B ( ϑ ) Γ ( ϑ + 3 ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ S B T 1 ( Ξ 1 ) 2 t o 1 1 ζ S B T 1 ( Ξ 2 ) + t o 2 1 η S B T 1 ( Ξ 3 ) ] Ψ ,

(56) I B T ( t δ + 1 ) = 1 ϑ A B ( ϑ ) t δ 1 ζ I B T 1 ( Ξ ) + ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ ) o = 2 δ I B T 1 ( Ξ 3 ) t o 2 1 ζ ϒ + ϑ ( Δ t ) ϑ A B ( ϑ ) Γ ( ϑ + 2 ) o = 2 δ 1 Δ t [ t o 1 1 ζ I B T 1 ( Ξ 2 ) t o 2 1 ζ I B T 1 ( Ξ 3 ) ] Φ + ϑ ( Δ t ) ϑ 2 A B ( ϑ ) Γ ( ϑ + 3 ) o = 2 δ 1 2 Δ t 2 [ t o 1 ζ I B T 1 ( Ξ 1 ) 2 t o 1 1 ζ I B T 1 ( Ξ 2 ) + t o 2 1 η I B T 1 ( Ξ 3 ) ] Ψ ,

where

(57) Ξ = ( t δ , S v ( t δ ) , I v ( t δ ) , S g ( t δ ) , I g ( t δ ) , S B T ( t δ ) , I B T ( t δ ) ) , Ξ 1 = ( t o , S v o , I v o , S g o , I g o , S B T o , I B T o ) , Ξ 2 = ( t o 1 , S v o 1 , I v o 1 , S g o 1 , I g o 1 , S B T o 1 , I B T o 1 ) , Ξ 3 = ( t o 2 , S v o 2 , I v o 2 , S g o 2 , I g o 2 , S B T o 2 , I B T o 2 ) , ϒ = [ ( δ o + 1 ) ϑ ( δ o ) ϑ ] , Φ = [ ( δ o + 1 ) ϑ ( δ o + 3 + 2 ϑ ) ( δ o ) ϑ ( δ o + 3 + 3 ϑ ) ] , Ψ = [ ( δ o + 1 ) ϑ { 2 ( δ o ) 2 + ( 3 ϑ + 10 ) ( δ o ) + 2 ϑ 2 + 9 ϑ + 12 } , ( δ o ) ϑ { 2 ( δ o ) 2 + ( 5 ϑ + 10 ) ( δ o ) + 6 ϑ 2 + 18 ϑ + 12 } ] .

Figure 1 
               Bayesian algorithm.
Figure 1

Bayesian algorithm.

5 Simulation and discussion

Using a variety of parameter settings and MATLAB methodologies, this section presents a fractal-fractional numerical simulation of a red chili plant virus model that efficiently solves fractal-fractional differential equations. Using initial points and parameter values from the study of Amelia et al. [47], the model was evaluated under various scenarios to find non-linear system results.

  • Figures 2, 3, 4, 5, 6, 7 show simulation data with different values of ϑ . Figures 27 demonstrate a graphical depiction of the red chili virus model using the suggested numerical approach with various values in the order of the fraction ϑ = 0.97,0.98,0.99 . We compare the results with the fractal-fractional number sequence in integer order. Each trajectory follows a similar pattern and leads to an actual endemic equilibrium point.

  • Figures 27 depict the dynamics of the plant and vector populations for a specific value of ϑ . The convergence rates of each graph (corresponding to ϑ ) differ, but all graph objects are in a stable state. Figure 2 shows that decreasing the order ϑ leads to an increase in the sensitive population of vegetative-phase plants.

  • Figure 3 shows that when the value of fractional-order ϑ decreases, the number of infected plants in the vegetative phase increases. Figure 4 shows that as ϑ drops, so does the vulnerable population of plants during the generative phase. Figure 5 shows that the value of ϑ drops as the infected plant population increases. Figure 6 shows that the vulnerable population of insect vectors increases as ϑ declines. Figure 7 shows that the infected population of insect vectors increases as the value of ϑ declines.

  • The graphical results demonstrate how effectively the target was met and how adding terms could potentially increase efficiency. To assess the influence of the fractional-order model, observations were made using fractional parameters. After decreasing fractions, solutions achieved the required reliability and precision. When compared to typical viral dynamic transmission in plants, the results show vector influence as well as the fractional derivative’s memory effect.

  • Figures 8, 9, 10, 11, 12, 13 provide a comparison of outcomes with the power law kernel (FFP) for fractional-order values of 0.99 and 0.95. Simulations are performed on all compartments to demonstrate the efficiency and rapid convergence to the steady-state point of the suggested approaches.

  • According to the study, the memory trace appears when fractional power increases from 0 to 1 and disappears at that moment. This memory effect makes advantage of a nonlinear proliferation that begins at zero to emphasize the distinction between integer and fractional-order derivatives. This confirms the occurrence of memory effects related to fractional-order derivatives.

  • Simulations demonstrate the important system parameters’ role in eradicating this viral infection in plants.

Figure 2 
               Model (simulation of proposed 
                     
                        
                        
                           
                              
                                 S
                              
                              
                                 v
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {S}_{v}\left(t)
                     
                  ) under FFM operator.
Figure 2

Model (simulation of proposed S v ( t ) ) under FFM operator.

Figure 3 
               Model (simulation of proposed 
                     
                        
                        
                           
                              
                                 I
                              
                              
                                 v
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {I}_{v}\left(t)
                     
                  ) under FFM operator.
Figure 3

Model (simulation of proposed I v ( t ) ) under FFM operator.

Figure 4 
               Model (simulation of proposed 
                     
                        
                        
                           
                              
                                 S
                              
                              
                                 g
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {S}_{g}\left(t)
                     
                  ) under FFM operator.
Figure 4

Model (simulation of proposed S g ( t ) ) under FFM operator.

Figure 5 
               Model (simulation of proposed 
                     
                        
                        
                           
                              
                                 I
                              
                              
                                 g
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {I}_{g}\left(t)
                     
                  ) under FFM operator.
Figure 5

Model (simulation of proposed I g ( t ) ) under FFM operator.

Figure 6 
               Model (simulation of proposed 
                     
                        
                        
                           
                              
                                 S
                              
                              
                                 B
                                 T
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {S}_{BT}\left(t)
                     
                  ) under FFM operator.
Figure 6

Model (simulation of proposed S B T ( t ) ) under FFM operator.

Figure 7 
               Model (simulation of proposed 
                     
                        
                        
                           
                              
                                 I
                              
                              
                                 B
                                 T
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {I}_{BT}\left(t)
                     
                  ) under FFM operator.
Figure 7

Model (simulation of proposed I B T ( t ) ) under FFM operator.

Figure 8 
               Model (comparison of 
                     
                        
                        
                           
                              
                                 S
                              
                              
                                 v
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {S}_{v}\left(t)
                     
                   with FFP and FFM operator) at fractional-order 0.99 and 0.95.
Figure 8

Model (comparison of S v ( t ) with FFP and FFM operator) at fractional-order 0.99 and 0.95.

Figure 9 
               Model (comparison of 
                     
                        
                        
                           
                              
                                 I
                              
                              
                                 v
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {I}_{v}\left(t)
                     
                   with FFP and FFM operator) at fractional-order 0.99 and 0.95.
Figure 9

Model (comparison of I v ( t ) with FFP and FFM operator) at fractional-order 0.99 and 0.95.

Figure 10 
               Model (comparison of 
                     
                        
                        
                           
                              
                                 S
                              
                              
                                 g
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {S}_{g}\left(t)
                     
                   with FFP and FFM operator) at fractional-order 0.99 and 0.95.
Figure 10

Model (comparison of S g ( t ) with FFP and FFM operator) at fractional-order 0.99 and 0.95.

Figure 11 
               Model (comparison of 
                     
                        
                        
                           
                              
                                 I
                              
                              
                                 g
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {I}_{g}\left(t)
                     
                   with FFP and FFM operator) at fractional-order 0.99 and 0.95.
Figure 11

Model (comparison of I g ( t ) with FFP and FFM operator) at fractional-order 0.99 and 0.95.

Figure 12 
               Model (comparison of 
                     
                        
                        
                           
                              
                                 S
                              
                              
                                 B
                                 T
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {S}_{BT}\left(t)
                     
                   with FFP and FFM operator) at fractional-order 0.99 and 0.95.
Figure 12

Model (comparison of S B T ( t ) with FFP and FFM operator) at fractional-order 0.99 and 0.95.

Figure 13 
               Model (comparison of 
                     
                        
                        
                           
                              
                                 I
                              
                              
                                 B
                                 T
                              
                           
                           
                              (
                              
                                 t
                              
                              )
                           
                        
                        {I}_{BT}\left(t)
                     
                   with FFP and FFM operator) at fractional-order 0.99 and 0.95.
Figure 13

Model (comparison of I B T ( t ) with FFP and FFM operator) at fractional-order 0.99 and 0.95.

6 Conclusion

This study looks into the economic and environmental effects of plant infections, specifically the fractional-order mathematical model of yellow virus transmission in C. annuum, which can result in lower crop yields, increased production costs, and the need for pesticide and control measures. The model was analyzed quantitatively and qualitatively to ensure bounded and positive solutions. The model’s existence and uniqueness were established by global impacts, and its local stability was assessed using Lipschitz and linear growth processes. The global stability was confirmed with the Lyapunov function. The impact of fractional operators on plant disease was investigated using a two-level Lagrange polynomial with the help of a Mittag–Leffler kernel. Numerical simulations are used to validate the model’s behavior, which facilitates the development of mitigation strategies and improves understanding of the virus’s activity. Finally, for additional assistance, all theoretical conclusions are graphically illustrated using Matlab software. Real-time transmission structure computations make the model extremely reliable. A new modification proposes a summary of scenarios containing singularities or not. In contrast to the classical model, the fractional model includes a memory effect in the epidemic model. The fractional derivative with Mittag–Leffler kernel provides a more practical approach to the full disease transmission scenario. We employed numerical modeling to forecast red chilli plants disease dynamics based on a variety of characteristics. It demonstrates that mathematical modeling offers a unique perspective on these processes. Key factors have been established for efficient infection control and treatment. The fractional derivative investigates the disease’s spread from its starting place, whereas the integer derivative studies the situation at a single spot. The study shows that comparable approaches can be used to a variety of real-world situations, with the goal of informing future research.

Acknowledgments

The authors extend their appreciation to Prince Sattam bin Abdulaziz University for funding this research work through the Project Number (PSAU/2024/01/29080).

  1. Funding information: The authors extend their appreciation to Prince Sattam bin Abdulaziz University for funding this research work through the Project Number (PSAU/2024/01/29080).

  2. Author contributions: All authors have accepted responsibility for the entire content of this manuscript and approved its submission. K.S.N.: conceptualization, methodology, Investigation, writing – review and editing, formal analysis, software, writing – original draft. F.S.A.: formal analysis; writing – review and editing, validation. M.F.: conceptualization, methodology, investigation, writing – original draft, writing – review and editing, visualization.

  3. Conflict of interest: The authors state no conflict of interest.

  4. Data availability statement: All data generated or analyzed during this study are included in this published article.

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Received: 2024-09-03
Revised: 2025-03-18
Accepted: 2025-07-29
Published Online: 2025-09-24

© 2025 the author(s), published by De Gruyter

This work is licensed under the Creative Commons Attribution 4.0 International License.

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