Artikel
Open Access
Possible involvement of endoplasmic reticulum stress in the pathogenesis of Alzheimer’s disease
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Toru Hosoi
Veröffentlicht/Copyright:
19. Oktober 2015
Received: 2015-06-30
Accepted: 2015-09-01
Published Online: 2015-10-19
Published in Print: 2015-01-01
© 2015 Toru Hosoi et al.
Artikel in diesem Heft
- Endoplasmic reticulum stress response in the roadway for the effects of non-steroidal anti-inflammatory drugs
- Inhibition of kinase and endoribonuclease activity of ERN1/IRE1α affects expression of proliferation related genes in U87 glioma cells
- The unfolded protein response, inflammation, oscillators, and disease: a systems biology approach
- Saturated fatty acids induce endoplasmic reticulum stress in primary cardiomyocytes
- The role of proteotoxic stress in vascular dysfunction in the pathogenesis of Alzheimer’s disease
- From recordings of disulfide isomerases in action to reversal of maladaptive endoplasmic reticulum stress responses: proceedings on the ER & Redox Club Meeting held in Venice, April 2015
- Genesis of ER Stress in Huntington’s Disease
- Possible involvement of endoplasmic reticulum stress in the pathogenesis of Alzheimer’s disease
Schlagwörter für diesen Artikel
Alzheimer’s disease;
pathogenesis;
therapeutics development;
endoplasmic reticulum stress;
unfolded protein response
Creative Commons
BY-NC-ND 3.0
Artikel in diesem Heft
- Endoplasmic reticulum stress response in the roadway for the effects of non-steroidal anti-inflammatory drugs
- Inhibition of kinase and endoribonuclease activity of ERN1/IRE1α affects expression of proliferation related genes in U87 glioma cells
- The unfolded protein response, inflammation, oscillators, and disease: a systems biology approach
- Saturated fatty acids induce endoplasmic reticulum stress in primary cardiomyocytes
- The role of proteotoxic stress in vascular dysfunction in the pathogenesis of Alzheimer’s disease
- From recordings of disulfide isomerases in action to reversal of maladaptive endoplasmic reticulum stress responses: proceedings on the ER & Redox Club Meeting held in Venice, April 2015
- Genesis of ER Stress in Huntington’s Disease
- Possible involvement of endoplasmic reticulum stress in the pathogenesis of Alzheimer’s disease
