Artikel
Open Access
Inhibition of kinase and endoribonuclease activity of ERN1/IRE1α affects expression of proliferation related genes in U87 glioma cells
-
Oleksandr H. Minchenko
Veröffentlicht/Copyright:
25. Februar 2015
Received: 2014-08-20
Accepted: 2014-12-22
Published Online: 2015-02-25
Published in Print: 2015-01-01
© 2015 Oleksandr H. Minchenko et al.
Artikel in diesem Heft
- Endoplasmic reticulum stress response in the roadway for the effects of non-steroidal anti-inflammatory drugs
- Inhibition of kinase and endoribonuclease activity of ERN1/IRE1α affects expression of proliferation related genes in U87 glioma cells
- The unfolded protein response, inflammation, oscillators, and disease: a systems biology approach
- Saturated fatty acids induce endoplasmic reticulum stress in primary cardiomyocytes
- The role of proteotoxic stress in vascular dysfunction in the pathogenesis of Alzheimer’s disease
- From recordings of disulfide isomerases in action to reversal of maladaptive endoplasmic reticulum stress responses: proceedings on the ER & Redox Club Meeting held in Venice, April 2015
- Genesis of ER Stress in Huntington’s Disease
- Possible involvement of endoplasmic reticulum stress in the pathogenesis of Alzheimer’s disease
Schlagwörter für diesen Artikel
endoplasmic reticulum stress;
ERN1/IRE1α;
E2F8;
EPAS1;
HOXC6;
ATF3;
TBX3;
FOXF1;
U87 glioma
cells;
NHA/TS astrocyte cells
Creative Commons
BY-NC-ND 3.0
Artikel in diesem Heft
- Endoplasmic reticulum stress response in the roadway for the effects of non-steroidal anti-inflammatory drugs
- Inhibition of kinase and endoribonuclease activity of ERN1/IRE1α affects expression of proliferation related genes in U87 glioma cells
- The unfolded protein response, inflammation, oscillators, and disease: a systems biology approach
- Saturated fatty acids induce endoplasmic reticulum stress in primary cardiomyocytes
- The role of proteotoxic stress in vascular dysfunction in the pathogenesis of Alzheimer’s disease
- From recordings of disulfide isomerases in action to reversal of maladaptive endoplasmic reticulum stress responses: proceedings on the ER & Redox Club Meeting held in Venice, April 2015
- Genesis of ER Stress in Huntington’s Disease
- Possible involvement of endoplasmic reticulum stress in the pathogenesis of Alzheimer’s disease
