Article
Open Access
The role of proteotoxic stress in vascular dysfunction in the pathogenesis of Alzheimer’s disease
-
Ana Catarina R.G. Fonseca
Published/Copyright:
August 20, 2015
Received: 2014-11-26
Accepted: 2014-05-13
Published Online: 2015-08-20
Published in Print: 2015-01-01
© 2015 Ana Catarina R.G. Fonseca et al.
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Keywords for this article
Alzheimer‘s disease;
Aβ peptide;
endothelial
cells;
endoplasmic reticulum;
Unfolded Protein Response,;
calcium homeostasis;
redox homeostasis;
Ubiquitinproteasome
system, autophagy, apoptosis
Creative Commons
BY-NC-ND 3.0
Articles in the same Issue
- Endoplasmic reticulum stress response in the roadway for the effects of non-steroidal anti-inflammatory drugs
- Inhibition of kinase and endoribonuclease activity of ERN1/IRE1α affects expression of proliferation related genes in U87 glioma cells
- The unfolded protein response, inflammation, oscillators, and disease: a systems biology approach
- Saturated fatty acids induce endoplasmic reticulum stress in primary cardiomyocytes
- The role of proteotoxic stress in vascular dysfunction in the pathogenesis of Alzheimer’s disease
- From recordings of disulfide isomerases in action to reversal of maladaptive endoplasmic reticulum stress responses: proceedings on the ER & Redox Club Meeting held in Venice, April 2015
- Genesis of ER Stress in Huntington’s Disease
- Possible involvement of endoplasmic reticulum stress in the pathogenesis of Alzheimer’s disease
