Article
Open Access
Genesis of ER Stress in Huntington’s Disease
-
Marina Shenkman
Published/Copyright:
August 28, 2015
Received: 2015-06-08
Accepted: 2015-07-15
Published Online: 2015-08-28
Published in Print: 2015-01-01
© 2015 Marina Shenkman et al.
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- Endoplasmic reticulum stress response in the roadway for the effects of non-steroidal anti-inflammatory drugs
- Inhibition of kinase and endoribonuclease activity of ERN1/IRE1α affects expression of proliferation related genes in U87 glioma cells
- The unfolded protein response, inflammation, oscillators, and disease: a systems biology approach
- Saturated fatty acids induce endoplasmic reticulum stress in primary cardiomyocytes
- The role of proteotoxic stress in vascular dysfunction in the pathogenesis of Alzheimer’s disease
- From recordings of disulfide isomerases in action to reversal of maladaptive endoplasmic reticulum stress responses: proceedings on the ER & Redox Club Meeting held in Venice, April 2015
- Genesis of ER Stress in Huntington’s Disease
- Possible involvement of endoplasmic reticulum stress in the pathogenesis of Alzheimer’s disease
Keywords for this article
Huntington;
neurodegenerative disease;
conformational disease;
protein misfolding;
protein
aggregation;
unfolded protein response;
ER-associated
degradation
Creative Commons
BY-NC-ND 3.0
Articles in the same Issue
- Endoplasmic reticulum stress response in the roadway for the effects of non-steroidal anti-inflammatory drugs
- Inhibition of kinase and endoribonuclease activity of ERN1/IRE1α affects expression of proliferation related genes in U87 glioma cells
- The unfolded protein response, inflammation, oscillators, and disease: a systems biology approach
- Saturated fatty acids induce endoplasmic reticulum stress in primary cardiomyocytes
- The role of proteotoxic stress in vascular dysfunction in the pathogenesis of Alzheimer’s disease
- From recordings of disulfide isomerases in action to reversal of maladaptive endoplasmic reticulum stress responses: proceedings on the ER & Redox Club Meeting held in Venice, April 2015
- Genesis of ER Stress in Huntington’s Disease
- Possible involvement of endoplasmic reticulum stress in the pathogenesis of Alzheimer’s disease