Article
Open Access
Endoplasmic reticulum stress response in the roadway for the effects of non-steroidal anti-inflammatory drugs
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Fernanda L.B. Mügge
Published/Copyright:
February 24, 2015
Received: 2014-08-30
Accepted: 2014-12-15
Published Online: 2015-02-24
Published in Print: 2015-01-01
© 2015 Fernanda L.B. Mügge and Aristóbolo M. Silva
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- From recordings of disulfide isomerases in action to reversal of maladaptive endoplasmic reticulum stress responses: proceedings on the ER & Redox Club Meeting held in Venice, April 2015
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Keywords for this article
NSAIDs;
PERK;
IRE1;
ATF6;
CHOP;
GRP78;
XBP1;
cancer cells
Creative Commons
BY-NC-ND 3.0
Articles in the same Issue
- Endoplasmic reticulum stress response in the roadway for the effects of non-steroidal anti-inflammatory drugs
- Inhibition of kinase and endoribonuclease activity of ERN1/IRE1α affects expression of proliferation related genes in U87 glioma cells
- The unfolded protein response, inflammation, oscillators, and disease: a systems biology approach
- Saturated fatty acids induce endoplasmic reticulum stress in primary cardiomyocytes
- The role of proteotoxic stress in vascular dysfunction in the pathogenesis of Alzheimer’s disease
- From recordings of disulfide isomerases in action to reversal of maladaptive endoplasmic reticulum stress responses: proceedings on the ER & Redox Club Meeting held in Venice, April 2015
- Genesis of ER Stress in Huntington’s Disease
- Possible involvement of endoplasmic reticulum stress in the pathogenesis of Alzheimer’s disease