Startseite The obesity epidemic makes life difficult for patients with herniated lumbar discs – and for back-surgeons: Increased risk of complications
Artikel Öffentlich zugänglich

The obesity epidemic makes life difficult for patients with herniated lumbar discs – and for back-surgeons: Increased risk of complications

  • Frode Kolstad und Harald Breivik EMAIL logo
Veröffentlicht/Copyright: 1. Januar 2016
Veröffentlichen auch Sie bei De Gruyter Brill

In this issue of the Scandinavian Journal of Pain, Järvimäki et al. focus on problems and complications arising in pre-obese and obese patients who need back surgery, discectomy in particular [1].The epidemic of obesity has reached the Nordic countries, and for persons above 60 years of age, Western Europe has prevalence about 20% for men and about 30% for women [2].

Excess bodyweight is among the highest risk factors for increased burden of disease globally, contributing to development of ischaemic heart disease, hypertension, osteoarthritis, diabetes mellitus, and stroke, cancer of the colon, breast and endometrium [2]. In their survey of lumbar discectomy patients, Järvimäki et al. found that outcomes of surgery with respect to functional disability, depressed mood, and social activities were worse among those with BMI in the obese category (BMI >30kg/m2) [1]. Both the preobese (BMI >25 and <30) and the obese patients gained weight during the postoperative follow-up time of about 2 years.

It is almost self-evident that technical difficulties for the surgeon doing minimally invasive discectomies will be more severe when operating on an obese patient compare with a non-obese patient. This may be the reason for a higher prevalence of reopera-tions among their obese patients [1]. Postoperative complications and cost significantly increase compared with none-obese patients [3]. Similarly, the anaesthesia team will have more challenges anesthetizing an obese patient; turning an obese patient under full general anaesthesia to the prone position is also not a trivial undertaking. The increased costs are due to longer operating time, longer anaesthesia time, more often admission to the intensive care unit as well as longer hospital stay [3].

We therefore agree hole-heartily with Järvimäki et al. [1] that obese patients with prolapsed lumbar disc must be helped lose weight before elective surgery. It may even be easier to motivate the patient to do a serious attempt at losing weight when they can be promised a better outcome of a necessary operative discectomy.

After a successive discectomy, the reduced burden of pain must be exploited for a continuing weight loss and encouragement to increase their physical activity. An individualized and prolonged postoperative physiotherapy regimen should contribute significantly to motivation and success of physical training. This will ensure a longer benefit after their successful discectomy.

If the patients continue to gain weight, as the obese patients of Järvimäki did during the two years following their discectomy [1], and continue to remain physically passive, their overall risk of ill health, mental depression, and low quality of life will continue. If they cannot lose weight and they cannot be physically more active, the high risks of developing one or more of the serious chronic diseases (see above) looming in the future of obese patients will not be reduced by a discectomy [1, 2].


DOI of refers to article: http://dx.doi.org/10.1016/j.sjpain.2015.10.003.



Department of Pain Management and Research, Department of Anesthesiology, Oslo University Hospital, PB 4950 Nydalen, 0424 Oslo, Norway. Tel.: +47 23073691

  1. Conflict of interest: None declared.

References

[1] Järvimäki V, Kautiainen H, Haanpää M, Alahuhta S, Vakkala M. Obesity has an impact on outcome in lumbar disc surgery. Scand J Pain 2016;10:85–9.Suche in Google Scholar

[2] Haslam DW, James WP. Obesity. Lancet 2005;366:1197–209.Suche in Google Scholar

[3] Planchard RF, Higgins DM, Mallory GW, Puffer RC, Jacob JT, Curry TB, Kor DJ, Clarke MJ. The impact of obesity on perioperative resource utilization after elective spine surgery for degenerative disease. Glob Spine J 2015;5:287–93, http://dx.doi.org/10.1055/s-0035-1546819 [Epub 2015 March 4].Suche in Google Scholar

Published Online: 2016-01-01
Published in Print: 2016-01-01

© 2015 Scandinavian Association for the Study of Pain

Artikel in diesem Heft

  1. Editorial comment
  2. Plasma pro-inflammatory markers in chronic neuropathic pain: Why elevated levels may be relevant for diagnosis and treatment of patients suffering chronic pain
  3. Original experimental
  4. Plasma pro-inflammatory markers in chronic neuropathic pain: A multivariate, comparative, cross-sectional pilot study
  5. Editorial comment
  6. Genetic variability of pain – A patient focused end-point
  7. Observational study
  8. COMT and OPRM1 genotype associations with daily knee pain variability and activity induced pain
  9. Editorial comment
  10. Complex Regional Pain Syndrome (CRPS) after viper-bite in a pregnant young woman: Pathophysiology and treatment options
  11. Clinical pain research
  12. Complex regional pain syndrome following viper-bite
  13. Editorial comment
  14. An investigation into enlarging and reducing the size of mirror reflections of the hand on experimentally induced cold-pressor pain in healthy volunteers
  15. Original experimental
  16. An investigation into enlarging and reducing the size of mirror reflections of the hand on experimentally-induced cold-pressor pain in healthy human participants
  17. Editorial comment
  18. Multimodal Rehabilitation Programs (MMRP) for patients with longstanding complex pain conditions – The need for quality control with follow-up studies of patient outcomes
  19. Observational study
  20. Patients with chronic pain: One-year follow-up of a multimodal rehabilitation programme at a pain clinic
  21. Editorial comment
  22. Advancing methods for characterizing structure and functions of small nerve fibres in neuropathic conditions
  23. Clinical pain research
  24. Structural and functional characterization of nerve fibres in polyneuropathy and healthy subjects
  25. Editorial comment
  26. Stimulation-induced expression of immediate early gene proteins in the dorsal horn is increased in neuropathy
  27. Original experimental
  28. Stimulation-induced expression of immediate early gene proteins in the dorsal horn is increased in neuropathy
  29. Editorial comment
  30. Targeting glial dysfunction to treat post-surgical neuropathic pain
  31. Topical review
  32. Glial dysfunction and persistent neuropathic postsurgical pain
  33. Editorial comment
  34. Mechanisms of cognitive impairment in chronic pain patients can now be studied preclinically by inducing cognitive deficits with an experimental animal model of chronic neuropathic pain
  35. Original experimental
  36. Impaired recognition memory and cognitive flexibility in the ratL5–L6 spinal nerve ligation model of neuropathic pain
  37. Editorial comment
  38. Pain treatment with intrathecal corticosteroids: Much ado about nothing? But epidural corticosteroids for radicular pain is still an option
  39. Original experimental
  40. Analgesic properties of intrathecal glucocorticoids in three well established preclinical pain models
  41. Editorial comment
  42. The obesity epidemic makes life difficult for patients with herniated lumbar discs – and for back-surgeons: Increased risk of complications
  43. Observational study
  44. Obesity has an impact on outcome in lumbar disc surgery
  45. Editorial comment
  46. Finnish version of the fear-avoidance-beliefs questionnaire (FABQ) and the importance of validated questionnaires on FAB in clinical praxis and in research on low-back pain
  47. Clinical pain research
  48. Translation and validation of the Finnish version of the Fear-Avoidance Beliefs Questionnaire (FABQ)
  49. Editorial comment
  50. Pain, sleep and catastrophizing: The conceptualization matters Comment on Wilt JA et al. “A multilevel path model analysis of the relations between sleep, pain, and pain catastrophizing in chronic pain rehabilitation patients”
  51. Clinical pain research
  52. A multilevel path model analysis of the relations between sleep, pain, and pain catastrophizing in chronic pain rehabilitation patients
Heruntergeladen am 23.9.2025 von https://www.degruyterbrill.com/document/doi/10.1016/j.sjpain.2015.10.008/html
Button zum nach oben scrollen