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Mitochondria, redox signaling and cell death in cancer
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Simone Fulda
Published/Copyright:
June 10, 2016
Published Online: 2016-6-10
Published in Print: 2016-7-1
©2016 by De Gruyter
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Articles in the same Issue
- Frontmatter
- Guest Editorial
- Mitochondria, redox signaling and cell death in cancer
- HIGHLIGHT: EMBO WORKSHOP “MITOCHONDRIA, APOPTOSIS AND CANCER 2015”
- The anti-oxidant and pro-oxidant dichotomy of Bcl-2
- Mitosis and mitochondrial priming for apoptosis
- The role of Her2 and other oncogenes of the PI3K/AKT pathway in mitochondria
- Mechanisms of mitophagy: putting the powerhouse into the doghouse
- Controlling quality and amount of mitochondria by mitophagy: insights into the role of ubiquitination and deubiquitination
- Coordination of stress, Ca2+, and immunogenic signaling pathways by PERK at the endoplasmic reticulum
- Regulation of necroptosis signaling and cell death by reactive oxygen species
- Contrasting effects of cardiac glycosides on cisplatin- and etoposide-induced cell death
- iBH3: simple, fixable BH3 profiling to determine apoptotic priming in primary tissue by flow cytometry
- Review
- The impact of recent advances in genetics in understanding disease mechanisms underlying the long QT syndromes
Articles in the same Issue
- Frontmatter
- Guest Editorial
- Mitochondria, redox signaling and cell death in cancer
- HIGHLIGHT: EMBO WORKSHOP “MITOCHONDRIA, APOPTOSIS AND CANCER 2015”
- The anti-oxidant and pro-oxidant dichotomy of Bcl-2
- Mitosis and mitochondrial priming for apoptosis
- The role of Her2 and other oncogenes of the PI3K/AKT pathway in mitochondria
- Mechanisms of mitophagy: putting the powerhouse into the doghouse
- Controlling quality and amount of mitochondria by mitophagy: insights into the role of ubiquitination and deubiquitination
- Coordination of stress, Ca2+, and immunogenic signaling pathways by PERK at the endoplasmic reticulum
- Regulation of necroptosis signaling and cell death by reactive oxygen species
- Contrasting effects of cardiac glycosides on cisplatin- and etoposide-induced cell death
- iBH3: simple, fixable BH3 profiling to determine apoptotic priming in primary tissue by flow cytometry
- Review
- The impact of recent advances in genetics in understanding disease mechanisms underlying the long QT syndromes