Mitophagy: mechanisms, pathophysiological roles, and analysis
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Wen-Xing Ding
Dr. Wen-Xing Ding received his PhD from the National University of Singapore. He is currently an assistant professor at the Department of Pharmacology, Toxicology and Therapeutics at the University of Kansas Medical Center, USA. Dr. Ding’s Lab focuses on the mechanisms of mitophagy in alcohol and drug-induced liver injury. Their ultimate goal is to investigate the possibilities to attenuate alcohol and drug-induced liver injury by modulating autophagy.
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Xiao-Ming Yin
Dr. Xiao-Ming Yin, MD, PhD, is a Louis Y. Mazzini Professor of Pathology at the Department of Pathology and Laboratory Medicine, Indiana University School of Medicine. Dr. Yin’s research is centered on the mechanisms of apoptosis and autophagy in tissue injury and cancer therapy.
©2012 by Walter de Gruyter Berlin Boston
Articles in the same Issue
- Masthead
- Masthead
- Reviews
- Mitophagy: mechanisms, pathophysiological roles, and analysis
- Zinc networks: the cell-specific compartmentalization of zinc for specialized functions
- Cyclophilin involvement in the replication of hepatitis C virus and other viruses
- Minireviews
- TDP-43 in central nervous system development and function: clues to TDP-43-associated neurodegeneration
- Platelet function and thymosin β4
- Exportin T and Exportin 5: tRNA and miRNA biogenesis – and beyond
- Genes and Nucleic Acids
- Biological and molecular analysis of the pathogenic variant C3 of potato spindle tuber viroid (PSTVd) evolved during adaptation to chamomile (Matricaria chamomilla)
- Protein Structure and Function
- Heterologous production and functional and thermodynamic characterization of cation diffusion facilitator (CDF) transporters of mesophilic and hyperthermophilic origin
- Functional characterization of the Mycobacterium tuberculosis zinc metallopeptidase Zmp1 and identification of potential substrates
- Antimicrobial activity of human islet amyloid polypeptides: an insight into amyloid peptides’ connection with antimicrobial peptides
- Cell Biology and Signaling
- Necroptosis modulated by autophagy is a predominant form of melanoma cell death induced by sanguilutine
Articles in the same Issue
- Masthead
- Masthead
- Reviews
- Mitophagy: mechanisms, pathophysiological roles, and analysis
- Zinc networks: the cell-specific compartmentalization of zinc for specialized functions
- Cyclophilin involvement in the replication of hepatitis C virus and other viruses
- Minireviews
- TDP-43 in central nervous system development and function: clues to TDP-43-associated neurodegeneration
- Platelet function and thymosin β4
- Exportin T and Exportin 5: tRNA and miRNA biogenesis – and beyond
- Genes and Nucleic Acids
- Biological and molecular analysis of the pathogenic variant C3 of potato spindle tuber viroid (PSTVd) evolved during adaptation to chamomile (Matricaria chamomilla)
- Protein Structure and Function
- Heterologous production and functional and thermodynamic characterization of cation diffusion facilitator (CDF) transporters of mesophilic and hyperthermophilic origin
- Functional characterization of the Mycobacterium tuberculosis zinc metallopeptidase Zmp1 and identification of potential substrates
- Antimicrobial activity of human islet amyloid polypeptides: an insight into amyloid peptides’ connection with antimicrobial peptides
- Cell Biology and Signaling
- Necroptosis modulated by autophagy is a predominant form of melanoma cell death induced by sanguilutine
