Mutational Analyses of Connexin 26, Connexin 30 and Connexin 46 as Candidate Tumor Suppressor Genes in Parathyroid Carcinoma

J. Costa,; T.M. Shattuck,; Y. Imanishi,; N. Palanisamy,; R.D. Gaz,; D. Shoback,; O.H. Clark,; J.M. Monchik,; M.E. Wierman,; A. Hollenberg,; Κ. Tojo,; R.S.K. Chaganti,; A. Arnold,

doi:10.1515/IJDHD.2002.3.2.57

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Mutational Analyses of Connexin 26, Connexin 30 and Connexin 46 as Candidate Tumor Suppressor Genes in Parathyroid Carcinoma

J. Costa, , T.M. Shattuck, , Y. Imanishi, , N. Palanisamy, , R.D. Gaz, , D. Shoback, , O.H. Clark, , J.M. Monchik, , M.E. Wierman, , A. Hollenberg, , Κ. Tojo, , R.S.K. Chaganti, und A. Arnold,

Veröffentlicht/Copyright: 1. April 2002

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Aus der Zeitschrift International Journal on Disability and Human Development Band 3 Heft 2

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Costa,, J., Shattuck,, T.M., Imanishi,, Y., Palanisamy,, N., Gaz,, R.D., Shoback,, D., Clark,, O.H., Monchik,, J.M., Wierman,, M.E., Hollenberg,, A., Tojo,, Κ., Chaganti,, R.S.K. and Arnold,, A.. "Mutational Analyses of Connexin 26, Connexin 30 and Connexin 46 as Candidate Tumor Suppressor Genes in Parathyroid Carcinoma" International Journal on Disability and Human Development, vol. 3, no. 2, pp. 57-62. https://doi.org/10.1515/IJDHD.2002.3.2.57

Costa,, J., Shattuck,, T., Imanishi,, Y., Palanisamy,, N., Gaz,, R., Shoback,, D., Clark,, O., Monchik,, J., Wierman,, M., Hollenberg,, A., Tojo,, Κ., Chaganti,, R. & Arnold,, A. (). Mutational Analyses of Connexin 26, Connexin 30 and Connexin 46 as Candidate Tumor Suppressor Genes in Parathyroid Carcinoma. International Journal on Disability and Human Development, 3(2), 57-62. https://doi.org/10.1515/IJDHD.2002.3.2.57

Costa,, J., Shattuck,, T., Imanishi,, Y., Palanisamy,, N., Gaz,, R., Shoback,, D., Clark,, O., Monchik,, J., Wierman,, M., Hollenberg,, A., Tojo,, Κ., Chaganti,, R. and Arnold,, A. () Mutational Analyses of Connexin 26, Connexin 30 and Connexin 46 as Candidate Tumor Suppressor Genes in Parathyroid Carcinoma. International Journal on Disability and Human Development, Vol. 3 (Issue 2), pp. 57-62. https://doi.org/10.1515/IJDHD.2002.3.2.57

Costa,, J., Shattuck,, T.M., Imanishi,, Y., Palanisamy,, N., Gaz,, R.D., Shoback,, D., Clark,, O.H., Monchik,, J.M., Wierman,, M.E., Hollenberg,, A., Tojo,, Κ., Chaganti,, R.S.K. and Arnold,, A.. "Mutational Analyses of Connexin 26, Connexin 30 and Connexin 46 as Candidate Tumor Suppressor Genes in Parathyroid Carcinoma" International Journal on Disability and Human Development 3, no. 2 (): 57-62. https://doi.org/10.1515/IJDHD.2002.3.2.57

Costa, J, Shattuck, T, Imanishi, Y, Palanisamy, N, Gaz, R, Shoback, D, Clark, O, Monchik, J, Wierman, M, Hollenberg, A, Tojo, Κ, Chaganti, R, Arnold, A. Mutational Analyses of Connexin 26, Connexin 30 and Connexin 46 as Candidate Tumor Suppressor Genes in Parathyroid Carcinoma. International Journal on Disability and Human Development. ;3(2): 57-62. https://doi.org/10.1515/IJDHD.2002.3.2.57

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CONTENTS
The Cysteine-rich C-Flanking Region of the Thyrotropin Receptor has Very Ancient Phylogenetic Origins: Implications from Sequence Analysis
Mutational Analyses of Connexin 26, Connexin 30 and Connexin 46 as Candidate Tumor Suppressor Genes in Parathyroid Carcinoma
Inheritance of the Arg64 Polymorphic Variants of the β3-Adrenergic Receptor is Linked to Impaired Insulin Secretion
Results of Calcitonin Screening in a Central Upper Austrian Region
Multiple Endocrine Neoplasia 2B and an Undescended Thyroid Gland
BOOK REVIEWS. CALENDAR

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https://doi.org/10.1515/IJDHD.2002.3.2.57

Artikel in diesem Heft

CONTENTS
The Cysteine-rich C-Flanking Region of the Thyrotropin Receptor has Very Ancient Phylogenetic Origins: Implications from Sequence Analysis
Mutational Analyses of Connexin 26, Connexin 30 and Connexin 46 as Candidate Tumor Suppressor Genes in Parathyroid Carcinoma
Inheritance of the Arg64 Polymorphic Variants of the β3-Adrenergic Receptor is Linked to Impaired Insulin Secretion
Results of Calcitonin Screening in a Central Upper Austrian Region
Multiple Endocrine Neoplasia 2B and an Undescended Thyroid Gland
BOOK REVIEWS. CALENDAR