Toxicity of Alzheimer's disease-associated Aβ peptide is ameliorated in a Drosophila model by tight control of zinc and copper availability

Articles in the same Issue

1. REVIEW
2. Disruption of virus-host cell interactions and cell signaling pathways as an anti-viral approach against influenza virus infections
3. PROTEIN STRUCTURE AND FUNCTION
4. Conformational changes of the chaperone SecB upon binding to a model substrate – bovine pancreatic trypsin inhibitor (BPTI)
5. Iron-catalyzed oxidation of Trp residues in low-density lipoprotein
6. MOLECULAR MEDICINE
7. Influenza A virus infection activates cholesterol sulfotransferase (SULT2B1b) in the lung of female C57BL/6 mice
8. CELL BIOLOGY AND SIGNALING
9. Lack of ADAM15 in mice is associated with increased osteoblast function and bone mass
10. Improved lentiviral gene transfer into human embryonic stem cells grown in co-culture with murine feeder and stroma cells
11. Ligand-independent activation of the arylhydrocarbon receptor by ETK (Bmx) tyrosine kinase helps MCF10AT1 breast cancer cells to survive in an apoptosis-inducing environment
12. Insulin modulates glucose-dependent insulinotropic polypeptide (GIP) secretion from enteroendocrine K cells in rats
13. Toxicity of Alzheimer's disease-associated Aβ peptide is ameliorated in a Drosophila model by tight control of zinc and copper availability
14. PROTEOLYSIS
15. In-cell selectivity profiling of membrane-anchored and replicase-associated hepatitis C virus NS3-4A protease reveals a common, stringent substrate recognition profile
16. Analysis of the autoproteolytic activity of the recombinant helper component proteinase from zucchini yellow mosaic virus