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Corrigendum to: Genetic associations of leukoaraiosis indicate pathophysiological mechanisms in white matter lesions etiology
This erratum corrects the original online version which can be found here:
https://doi.org/10.1515/revneuro-2014-0082
-
Qing Lin
Published/Copyright:
August 12, 2015
Corrigendum to: Qing Lin, Wen-Qing Huang and Chi-Meng Tzeng, Genetic associations of leukoaraiosis indicate pathophysiological mechanisms in white matter lesions etiology. Rev. Neurosci. 26 (2015), pp. 343–358. DOI: 10.1515/revneuro-2014-0082.
During the proofreading process of the paper several inaccuracies escaped the authors’ attention and were transferred into the final article. The following list indicates the location and type of correction to the paper. The authors apologize for the inconvenience.
| Page | Text in the paper | Correction |
|---|---|---|
| 343, Introduction, 14th line from bottom | technique | techniques |
| 343, Introduction, 9th line from bottom | Parkinson’ | Parkinson’s |
| 344, Introduction, the first line in the last paragraph | focuses | focused |
| 346, Genetics of LA, 14th line from top | correlates | correlations |
| 348, left side, 28th line from top | WBP2 | WBP2 |
| 348, right side, 24th line from top | function | functions |
| 348, right side, 25th line from top | is | are |
| 348, right side, 26th line from top | needs | need |
| 348, right side, 27th line from top | paved | could pave |
| 349, left side, 25th line from top | these genes variants | these genes |
| 349, right side, 34th line from bottom | WM(L) | WM(C) |
| 349, right side, 31th line from bottom | genes of known function are | genes |
| 349, right side, 18th line from bottom | suggested | showed |
| 349, right side, 7th line from bottom | provides | provided |
| 349, right side, 1–2 lineth line from bottom | contribute to the pathogenesis of WML and are needed to search for the cause and as targets for future studies. | contribute to the pathogenesis of WML. |
| 350, right side, 23th line from top | inflammatory | inflammation |
| 350, right side, last line | contributes | may contribute |
| 351, left side, 23th line from bottom | the ischemia | the ischemia (or potential microbleeding) |
| 351, left side, 15th line from bottom | inflammation (neuroinflammatory) | inflammation |
| 351, left side, 14th line from bottom | coexist and play | coexisted and played |
| 351, left side, 12th line from bottom | also showed | showed |
| 351, left side, 2th line from bottom | plays | played |
| 351, right side, 12th–13th line from top | in response to the development of ischemia and to disturb the balance of (de)myelination | in response to the development of ischemia (or putative microbleeding) and distrubed the balance of (de)myelination |
| 351, right side, 19th line from bottom | Potential LA pathogenesis genes | Potential pathogenic genes of LA |
| 351, right side, 16th–17th line from bottom | as shown in Figure 2 under a complicated mechanism. | as shown in Figure 2. |
| 351, right side, 2th–3th line from bottom | investigating the correlation between genetic polymorphisms and mutations | investigating the correlation of genetic variants |
| 352, Figure 2, the third line from top | is considered as | are considered as |
| 352, Figure 2, the 8th line from top | in response to incomplete ischemia | in response to incomplete ischemia or microbleeding |
| 352, left side, the 5th line from bottom | and | with |
| 353, left side, the 11th line from top | dementia and stroke | dementia |
| 353, from the last line in the left side to the first line in the right side | as a WML neuroimaging in the elders (micro-bleeding and demyelination) but also implying caused by | as a WML neuroimaging in the elders but also implying microbleeding (or putative ischemia) and demyelination caused by |
| 353, right side, the third line from top | function | functions |
| 353, right side, the 4th line from top | with respect to | implicated in |
| 353, right side, the 11th line from top | biomarker | biomarkers |
| 358, Bionotes (Qing Lin), left side, the 7th line from bottom | Master’s | Master |
| 358, Bionotes (Qing Lin), left side, the 5th line from bottom | lies | lay |
| 358, Bionotes (Qing Lin), right side, the third line from top | identifying idiopathic normal pressure | idiopathic normal pressure |
| 358, Bionotes (Wen-Qing Huang), right side, the 10th line from top | collaborating with The First Affiliated Hospital of Xiamen University | collaborating with Dr. Lin from The First Affiliated Hospital of Xiamen University |
| 358, Bionotes (Chi-Meng Tzeng), right side, from the first line to the fourth line | Chi-Meng Tzeng received comprehensive graduate training in Tsinhua University (Radiation Biology, advised by Dr. Rong-Long Pan) and Stanford University (Medical School in Biochemistry, advised by Dr. Arthur Kornberg) in the 1990s. | Chi-Meng Tzeng received comprehensive graduate training in the College of Nuclear Science in National Tsing Hua University (Radiation Biology, advised by Dr. Rong-Long Pan) and Stanford University (Structural biology and Biochemistry, advised by Dr. Peter Parham and Dr. Arthur Kornberg) in the 1990s. |
| 358, Bionotes (Chi-Meng Tzeng), right side, 9th–10th line from bottom | Center of Translational Medicine Research, Institute for | Translational Medicine Research Center (TMRC) |
| 358, Bionotes (Chi-Meng Tzeng), right side, 8th line from bottom | College of Pharmacy | School of Pharmaceutical Sciences |
| 358, Bionotes (Chi-Meng Tzeng), right side, 3th–4th line from bottom | and leukoaraiosis (genetic diagnosis or prognosis). | leukoaraiosis (genetic diagnosis or prognosis) and cell therapy (T-cell and MSC). |
| 358, Bionotes (Chi-Meng Tzeng), right side, the last line | new drug applications | new drug or cancer vaccination applications |
Published Online: 2015-8-12
Published in Print: 2015-10-1
©2015 by De Gruyter
Articles in the same Issue
- Frontmatter
- On the role of the extracellular space on the holistic behavior of the brain
- Temporal phases of long-term potentiation (LTP): myth or fact?
- Are there treatments for atypical parkinsonism? An update on actual options
- Clinical experimental stress studies: methods and assessment
- Genetic aspects of intervertebral disc degeneration
- Corrigendum
- Corrigendum to: Genetic associations of leukoaraiosis indicate pathophysiological mechanisms in white matter lesions etiology
Articles in the same Issue
- Frontmatter
- On the role of the extracellular space on the holistic behavior of the brain
- Temporal phases of long-term potentiation (LTP): myth or fact?
- Are there treatments for atypical parkinsonism? An update on actual options
- Clinical experimental stress studies: methods and assessment
- Genetic aspects of intervertebral disc degeneration
- Corrigendum
- Corrigendum to: Genetic associations of leukoaraiosis indicate pathophysiological mechanisms in white matter lesions etiology
